Phenotype specific analyses of p53 reveal distinct regulatory mechanism for chronically activated p53

ABSTRACT: p53 ChIP seq and Histone mark ChIP Seq data in IMR90 (normal human diploid fibroblasts) Phenotypes were created by overexpression of RasG12V, E1A/RasG12V in IMR90 cells, growing IMR90 and 24 hour Etoposide treated IMR90 cells were subjected to p53 and histone mark ChIP Seq

ORGANISM(S): Homo sapiens

SUBMITTER: Silvia Halim 

PROVIDER: E-GEOD-53491 | biostudies-arrayexpress |

REPOSITORIES: biostudies-arrayexpress