BORIS/CTCFL reprograms glioblastoma transcriptional networks through regulation of tumor-associated genes such as CD36 and FBN2

ABSTRACT: Glioblastoma multiforme (GBM) remains one of the most aggressive and lethal brain tumors. BORIS (CTCFL) functions as an epigenetic and transcriptional regulator implicated in multiple cancers; however, its role in GBM is poorly understood. To explore its regulatory function, we performed ChIP-seq profiling in U87MG cells.

INSTRUMENT(S): Illumina NovaSeq 6000

ORGANISM(S): Homo sapiens

SUBMITTER: Gerardo Ramirez-Mejia 

PROVIDER: E-MTAB-15935 | biostudies-arrayexpress |

REPOSITORIES: biostudies-arrayexpress