BORIS/CTCFL reprograms glioblastoma transcriptional networks through regulation of tumor-associated genes such as CD36 and FBN2
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ABSTRACT: Glioblastoma multiforme (GBM) remains one of the most aggressive and lethal brain tumors. BORIS (CTCFL) functions as an epigenetic and transcriptional regulator implicated in multiple cancers; however, its role in GBM is poorly understood. To explore its regulatory function, we performed ChIP-seq profiling in U87MG cells.
INSTRUMENT(S): Illumina NovaSeq 6000
ORGANISM(S): Homo sapiens
SUBMITTER: Gerardo Ramirez-Mejia
PROVIDER: E-MTAB-15935 | biostudies-arrayexpress |
REPOSITORIES: biostudies-arrayexpress
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