Metabolic and transcriptional response of Arabidopsis thaliana wildtype and mutants to Phytophtora infestans

ABSTRACT: The oomycete P. infestans is the causal agent of late blight, the most devastating potato disease. In contrast to potato, A. thaliana is able to successfully prevent colonization of the pathogen due to a multi-layered nonhost resistance. Several mutants have been isolated which are impaired in penetration resistance. A mutation in the gene PEN2, which encodes for an enzyme involved in indole glucosinolate metabolism (Bednarek et al. (2009)), results in the loss of penetration resistance against P. infestans (Lipka et al. (2005)). Despite its ability to penetrate epidermal cells of pen2 mutant plants, P. infestans is still not able to colonize these plants. Additional mutants were isolated by Kopischke et al. (2013) which show enhanced defense responses upon infection with P. infestans: pen

INSTRUMENT(S): 418 [Affymetrix]

ORGANISM(S): Arabidopsis thaliana

SUBMITTER: Steffen Neumann 

PROVIDER: E-MTAB-3287 | biostudies-arrayexpress |

REPOSITORIES: biostudies-arrayexpress