Oncogenic GNAS Uses PKA-Dependent and Independent Mechanisms to Induce Cell Proliferation in Human Pancreatic Ductal and Acinar Organoids.

ABSTRACT:

Implications

The study identifies an opportunity to discover a PKA-independent pathway downstream of oncogene GNAS for managing IPMN lesions and their progression to PDAC.

SUBMITTER: Desai R 

PROVIDER: S-EPMC10906748 | biostudies-literature | 2024 May

REPOSITORIES: biostudies-literature