Neurophysiological insights into sit-to-stand post stroke.

ABSTRACT:

Introduction

Stroke often results in the loss of ability to stand-up independently or to perform the transfer with compensatory movement patterns. While neurological disorders are associated with sit-to-stand disability, the neurophysiological mechanisms underlying the movement and the impact of injury at brain level remain poorly understood.

Methods

Stroke participants (n = 10, 4 males) performed five sets of three sit-to-stand transitions from an armless, backless seat adjusted to their knee joint height with three-dimensional kinematic data capture. Electromyography (EMG) was recorded from the bilateral vastus lateralis, biceps femoris, tibialis anterior, and gastrocnemius muscles. Surface electroencephalography (EEG) activity was recorded using eight focused bipol

SUBMITTER: McDonald C 

PROVIDER: S-EPMC12436286 | biostudies-literature | 2025

REPOSITORIES: biostudies-literature