Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naive non-transgenic mice induces robust α-synuclein pathology.

ABSTRACT:

SUBMITTER: Sacino AN 

PROVIDER: S-EPMC4852859 | biostudies-literature | 2016 Jan

REPOSITORIES: biostudies-literature