Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naive non-transgenic mice induces robust α-synuclein pathology.
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ABSTRACT:
SUBMITTER: Sacino AN
PROVIDER: S-EPMC4852859 | biostudies-literature | 2016 Jan
REPOSITORIES: biostudies-literature
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Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology.
Sacino Amanda N AN Ayers Jacob I JI Brooks Mieu M T MM Chakrabarty Paramita P Hudson Vincent J VJ Howard Jasie K JK Golde Todd E TE Giasson Benoit I BI Borchelt David R DR
Acta neuropathologica 20151105 1
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