Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naive non-transgenic mice induces robust α-synuclein pathology.
Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology.