Estradiol stimulates glycogen synthesis whereas progesterone promotes glycogen catabolism in the uterus of the American mink (Neovison vison).
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ABSTRACT: Glycogen synthesis by mink uterine glandular and luminal epithelia (GE and LE) is stimulated by estradiol (E2 ) during estrus. Subsequently, the glycogen deposits are mobilized to near completion to meet the energy requirements of pre-embryonic development and implantation by as yet undetermined mechanisms. We hypothesized that progesterone (P4 ) was responsible for catabolism of uterine glycogen reserves as one of its actions to ensure reproductive success. Mink were treated with E2 , P4 or vehicle (controls) for 3 days and uteri collected 24 h (E2 , P4 and vehicle) and 96 h (E2 ) later. To evaluate E2 priming, mink were treated with E2 for 3 days, then P4 for an additional 3 days
SUBMITTER: Bowman K
PROVIDER: S-EPMC5107167 | biostudies-literature | 2017 Jan
REPOSITORIES: biostudies-literature
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