Anilinopyrimidine Resistance in Botrytis cinerea Is Linked to Mitochondrial Function.

ABSTRACT: Crop protection anilinopyrimidine (AP) fungicides were introduced more than 20 years ago for the control of a range of diseases caused by ascomycete plant pathogens, and in particular for the control of gray mold caused by Botrytis cinerea. Although early mode of action studies suggested an inhibition of methionine biosynthesis, the molecular target of this class of fungicides was never fully clarified. Despite AP-specific resistance having been described in B. cinerea field isolates and in multiple other targeted species, the underlying resistance mechanisms were unknown. It was therefore expected that the genetic characterization of resistance mechanisms would permit the identification of the molecular target of these fungicides. In order to explore the widest range

SUBMITTER: Mosbach A 

PROVIDER: S-EPMC5714876 | biostudies-literature | 2017

REPOSITORIES: biostudies-literature