ABA-mediated regulation of stomatal density is OST1-independent.
Ontology highlight
ABSTRACT: Stomata, small pores on the surfaces of leaves formed by a pair of guard cells, adapt rapidly to changes in the environment by adjusting the aperture width. As a long-term response, the number of stomata is regulated during stomatal development. The hormone abscisic acid (ABA) regulates both processes. In ABA mediated guard cell signaling the protein kinase OPEN STOMATA1 (OST1) has a central role, as stomatal closure in the ost1 mutant is impaired in response to ABA and to different environmental stimuli. We aimed to dissect the contribution of different ABA-related regulatory mechanisms in determining stomatal conductance, a combination of stomatal density and aperture width, and crossed the ost1 mutant with mutants that either decreased (aba3) or increased (cyp707a1/a3) the concentration
SUBMITTER: Jalakas P
PROVIDER: S-EPMC6508810 | biostudies-literature | 2018 Sep
REPOSITORIES: biostudies-literature
ACCESS DATA