Hippocampal mGluR1-dependent long-term potentiation requires NAADP-mediated acidic store Ca<sup>2+</sup> signaling.
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ABSTRACT: Acidic organelles, such as endosomes and lysosomes, store Ca2+ that is released in response to intracellular increases in the second messenger nicotinic acid adenine dinucleotide phosphate (NAADP). In neurons, NAADP and Ca2+ signaling contribute to synaptic plasticity, a process of activity-dependent long-term potentiation (LTP) [or, alternatively, long-term depression (LTD)] of synaptic strength and neuronal transmission that is critical for neuronal function and memory formation. We explored the function of and mechanisms regulating acidic Ca2+ store signaling in murine hippocampal neurons. We found that metabotropic glutamate receptor 1 (mGluR1) was coupled to NAADP signaling that elicited Ca2+ release from acidic stores. In turn, this release
SUBMITTER: Foster WJ
PROVIDER: S-EPMC6679716 | biostudies-literature | 2018 Nov
REPOSITORIES: biostudies-literature
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