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The Ca²⁺-CaM Signaling Pathway Mediates Potassium Uptake by Regulating Reactive Oxygen Species Homeostasis in Tobacco Roots Under Low-K⁺ Stress.

ABSTRACT: Potassium (K⁺) deficiency severely threatens crop growth and productivity. Calcium (Ca²⁺) signaling and its sensors play a central role in the response to low-K⁺ stress. Calmodulin (CaM) is an important Ca²⁺ sensor. However, the mechanism by which Ca²⁺ signaling and CaM mediate the response of roots to low-K⁺ stress remains unclear. In this study, we found that the K⁺ concentration significantly decreased in both shoots and roots treated with Ca²⁺ channel blockers, a Ca²⁺ chelator, and CaM antagonists. Under low-K⁺ stress, reactive oxygen species (ROS) accumulated, and the activity of antioxidant enzymes, NAD kinase (NADK), and NADP phosphatase (NADPase) decreased. This indicates t

SUBMITTER: Wang Y

PROVIDER: S-EPMC8216240 | biostudies-literature | 2021

REPOSITORIES: biostudies-literature

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The Ca²⁺-CaM Signaling Pathway Mediates Potassium Uptake by Regulating Reactive Oxygen Species Homeostasis in Tobacco Roots Under Low-K⁺ Stress.

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The Ca2+-CaM Signaling Pathway Mediates Potassium Uptake by Regulating Reactive Oxygen Species Homeostasis in Tobacco Roots Under Low-K+ Stress.

OmicsDI is part of the ELIXIR infrastructure

Tweets

The Ca²⁺-CaM Signaling Pathway Mediates Potassium Uptake by Regulating Reactive Oxygen Species Homeostasis in Tobacco Roots Under Low-K⁺ Stress.