Nitric oxide promotes energy metabolism and protects mitochondrial DNA in peaches during cold storage.

ABSTRACT: The mitochondria are important organelles related to energy metabolism and are susceptible to oxidative damage. In this experiment, peaches (Prunus persica) were treated with distilled water (as the control), 15 μmol L^-1 of nitric oxide (NO), and 20 μmol L^-1 of carboxy-PTIO (NO scavenger). The changes in mitochondrial physiological indicators, energy metabolism process, and mitochondrial DNA (mtDNA) damage and repair were quantified. Compared with the control, NO treatment reduced mitochondrial oxygen consumption and the reactive oxygen species content, increased mitochondrial respiration control rate, and promoted energy metabolism by influencing the activities of citrate synthase, aconitase, isocitrate dehydrogenase, and α-ketoglutarate dehydrogenase in the t

SUBMITTER: Ren Y 

PROVIDER: S-EPMC9582448 | biostudies-literature | 2022

REPOSITORIES: biostudies-literature