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ABSTRACT: Background
The annual yield losses caused by the Rice Blast Fungus, Magnaporthe oryzae, range to the equivalent for feeding 60 million people. To ward off infection by this fungus, rice has evolved a generic basal immunity (so called compatible interaction), which acts in concert with strain-specific defence (so-called incompatible interaction). The plant-defence hormone jasmonic acid (JA) promotes the resistance to M. oryzae, but the underlying mechanisms remain elusive. To get more insight into this open question, we employ the JA-deficient mutants, cpm2 and hebiba, and dissect the JA-dependent defence signalling in rice for both, compatible and incompatible interactions.Results
We observe that both JA-deficient mutants are more susceptible to M. oryzae as compared to the
SUBMITTER: Ma J
PROVIDER: S-EPMC9764487 | biostudies-literature | 2022 Dec
REPOSITORIES: biostudies-literature