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Expression data from P4H-TM deficient mouse cortexes after 24h of pMCAO

ABSTRACT: Genetic deletion of transmembrane prolyl-4-hydroxylase (P4H-TM) in mice leads to increased inflammatory microgliosis and neutrophil infiltration in the cortex after permanent midle cerebral artery occlusion (pMCAO)

ORGANISM(S): Mus musculus

PROVIDER: GSE107127 | GEO | 2020/11/12

REPOSITORIES: GEO

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mRNA profiles after experimental ischemia

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