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PU.1 ChIP-seq time course in mouse acute myeloid leukemia cells upon PU.1 restoration and knockdown

ABSTRACT: We have utilized advanced RNAi technology to dynamically restore and subsequently knock down endogenous PU.1 expression in p53-deficient AML cells, which respectively causes differentiation and de-differentiation

ORGANISM(S): Mus musculus

PROVIDER: GSE108945 | GEO | 2019/05/13

REPOSITORIES: GEO

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