The alterations of gene expression in aorta from endothelial nitric oxide synthase (eNOS)-deficient mice

ABSTRACT: The dysfunction of endothelial nitric oxide synthase may be involved in development of atherosclerosis; however, the underlying molecular and cellular mechanisms of atherosclerosis are poorly understood. Here, we investigated gene expressionsin relation to atherosclerosis using endothelial nitric oxide synthase (eNOS)-deficient mice.

ORGANISM(S): Mus musculus

PROVIDER: GSE123855 | GEO | 2018/12/15

REPOSITORIES: GEO