Glomerular endothelial cell and podocyte stresses as well as crosstalk in structurally normal kidney transplants

ABSTRACT: Even among apparently healthy human allografts with no detectable histologic abnormality, transcriptomic changes reflecting cell stresses have already been set in motion that could drive hypertrophy-associated glomerular disease progression

ORGANISM(S): Homo sapiens

PROVIDER: GSE169285 | GEO | 2021/12/23

REPOSITORIES: GEO