Loss of BAP1 cooperates with TP53 deficiency to transform a common myelo-erythroid progenitor (ChIP-seq - mus musculus)
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ABSTRACT: We performed RNA-seq and ChIP-seq in lineage depleted bone marrow cells isolated from single and double Bap1 and Trp53 knockout mice to identify transcriptional and epigenetic programs that drive erythroleukemia.
ORGANISM(S): Mus musculus
PROVIDER: GSE173547 | GEO | 2026/03/05
REPOSITORIES: GEO
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