HuR promotes castration-resistant prostate cancer progression by altering ERK5 activation via posttranscriptional regulation of BCAT1
Ontology highlight
ABSTRACT: Castration-resistant prostate cancer (CRPC) is refractory to hormone treatment, and its progression mechanism is not fully uncovered. This study aims to clarify the role and mechanism of Human antigen R (HuR) as a therapeutic target for CRPC progression.
ORGANISM(S): Homo sapiens
PROVIDER: GSE254851 | GEO | 2024/02/05
REPOSITORIES: GEO
ACCESS DATA