Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

RNA-seq of DLX6 KD A549 cells vs. control A549 cells.

ABSTRACT: To investigate the gene expression characteristics of the transcription factor DLX6, we performed RNA-seq analysis in A549 cells to examine changes in the gene expression profile following DLX6 knockdown.

ORGANISM(S): Homo sapiens

PROVIDER: GSE294066 | GEO | 2026/04/14

REPOSITORIES: GEO

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
		Other

Items per page:

1 - 1 of 1

Similar Datasets

RNA-seq of DLX6 KD A549 cells vs. control A549 cells.

Project description:RNA-seq of DLX6 KD A549 cells vs. control A549 cells.

| PRJNA1247983 | ENA

RNA-seq of UHRF1 KO A549 cells vs. control A549 cells.

Project description:RNA-seq of UHRF1 KO A549 cells vs. control A549 cells.

| PRJNA1119458 | ENA

A549 treated with CISPLATIN/TSA VS CONTROL

Project description:WE TEST THE PROTEIN CHANGES WHEN A549 cells treated with DDP OR TSA.

2024-06-23 | PXD052305 | Pride

RNA-seq analysis using A549 cells with TRIM22 knockdown and control A549 cells with or without ionizing radiation treatment.

Project description:In this study, we explored the role of TRIM22 in the response to ionizing radiation (IR) in A549 human lung cancer cells. TRIM22 was stably knocked down in A549 cells using shRNA, and four experimental groups were established: control A549 cells (non-irradiated), control A549 cells (irradiated), TRIM22 knockdown A549 cells (non-irradiated), and TRIM22 knockdown A549 cells (irradiated). RNA sequencing (RNA-seq) was performed to identify gene expression changes associated with TRIM22 knockdown and irradiation. This dataset provides insights into the molecular mechanisms by which TRIM22 influences the cellular response to radiation and its potential impact on cancer therapy.

2025-01-01 | GSE277805 | GEO

RNA-seq analysis using A549 cells with FXR knockdown and control A549 cells with or without ionizing radiation treatment.

Project description:In this study, we explored the role of FXR in the response to ionizing radiation (IR) in A549 human lung cancer cells. FXR was stably knocked down in A549 cells using shRNA, and four experimental groups were established: control A549 cells (non-irradiated), control A549 cells (irradiated), FXR knockdown A549 cells (non-irradiated), and FXR knockdown A549 cells (irradiated). RNA sequencing (RNA-seq) was performed to identify gene expression changes associated with FXR knockdown and irradiation. This dataset provides insights into the molecular mechanisms by which FXR influences the cellular response to radiation and its potential impact on cancer therapy.

2024-10-31 | GSE278872 | GEO

A549 MMAB knockdown

Project description:A549 MMAB knockdown

| PRJEB100796 | ENA

A549 cells and MSR-A549 cells

Project description:A549 cells and MSR-A549 cells

| PRJNA379619 | ENA

Secretome analysis of HSPD1 knockdown in A549 lung cancer cells

Project description:HSPD1 knockdown was established in A549 cells using an shRNA approach. Secretomes from both shHSPD1-A549 and shControl-A549 cells were collected and subjected to label-free comparative proteome analysis using SWATH-MS.

2024-04-13 | PXD050755 | Pride

Long term radiation effects in A549 cells - PML knock down and carbon ion vs. photon irradiation

Project description:The aim of our study is to investigate the effects of carbon ion and photon irradiation on A549 tumor cells and analyse how these effects are altered by PML knockdown. Therefore we created PML knockdown A549 cells (shPML) and irradiated them with either 2Gy carbon ion or 6Gy Photon (bioequivalent doses). 4 days after irradiation microarray analysis was performed. All experiments were performed in 3 biological replicates and control groups were transduced with an empty vector.

2023-11-02 | E-MEXP-3965 | biostudies-arrayexpress

RNPC3 knockdown in A549 lung adenocarcinoma cells

Project description:RNPC3 knockdown in A549 lung adenocarcinoma cells

| PRJNA789044 | ENA