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Human gastric cancer cells SGC7901-NM RIP-Chip result: has-miR-625 lentivirus infected vs. control

ABSTRACT: Transcriptional profiling of RIP products of human gastric cancer cells SGC7901-NM comparing control with SGC7901-NM infected with has-miR-625 lentivirus

ORGANISM(S): Homo sapiens

PROVIDER: GSE38811 | GEO | 2012/06/20

SECONDARY ACCESSION(S): PRJNA169030

REPOSITORIES: GEO

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Human gastric cancer cells SGC7901-NM RIP-Chip result: has-miR-625 lentivirus infected vs. control

Project description:Transcriptional profiling of RIP products of human gastric cancer cells SGC7901-NM comparing control with SGC7901-NM infected with has-miR-625 lentivirus Stable transfected cell lines, SGC7901-NM-has-miR-625 vs. SGC7901-NM-NC, after RNA-binding protein immunoprecipitation with Ago2 antibody, the experimental group (Ago2) vs. the control group (input) per array.

2012-06-19 | E-GEOD-38811 | biostudies-arrayexpress

Homo sapiens

Project description:Human gastric cancer cells SGC7901-NM RIP-Chip result: has-miR-625 lentivirus infected vs. control

| PRJNA169030 | ENA

Next generation sequencing analysis of mRNA profile in cisplatin-resistant gastric cancer cell line SGC7901

Project description:Cisplatin-resistant gastric cancer (GC) occurs in patients with GC treated with cisplatin-based chemotherapy, which results in disease progression and early recurrence during the treatment. To understand the initiation and developmental mechanism underlying cisplatin-resistant GC, we developed cisplatin resistant SGC7901 cells (SGC7901/DDP) from the parental cells (SGC7901/S) by continuous exposure to increasing concentrations of cisplatin and subjected these two cell lines to RNA sequencing analysis.

2019-04-17 | GSE122130 | GEO

Genome-wide analysis of gene expression by SGC7901 subjected to recombinant human STIP1 (rhSTIP1)

Project description:Analysis of SGC7901 in proliferation, apoptosis, metastasis, and metabolism at gene expression level. The hypothesis tested in the present study was that rhSTIP1 influences the epithelial-to-mesenchymal transition of SGC7901. Results provide important information of the epithelial-to-mesenchymal transition, such as Wnt signaling pathway genes, up- or down-regulated specific metastasis cellular functions.

2018-02-14 | GSE107819 | GEO

Streptococcus sp. NM strain:NM

Project description:Streptococcus sp. NM strain:NM Genome sequencing and assembly

| PRJNA484102 | ENA

Identify METTL3, 14 and 16-bound transcripts via RIP-seq

Project description:To identify the directly bound transcripts of METTL3, METTL14, and METTL16, RNA immunoprecipitation sequencing (RIP-seq) was conducted HEK293T stablely expressing METTL3, METTL14, and METTL16, respectively. Briefly, HEK293T cells were infected with lentivirus, pmiRNA1-3 x Flag-METTL3, pmiRNA1-3 x Flag-METTL14, and pmiRNA1-3 x Flag-METTL14, to overexpress the three METTL family members with 3 x Flag fused in the N-terminal. Only the GFP-positive cells were used for study and expanded in DMEM medium.

2021-10-28 | GSE156797 | GEO

The RNA-seq result of GC cells which were treated with chrysin

Project description:Chrysin has been reported to have anticancer effect. We used 40μM chrysin to treat SGC7901 cells for 48h, then sequenced their RNA, analyzed the differentially expressed genes before and after treatment, screened out the anticancer target of chrysin, and then carried on the next analysis

2021-08-19 | GSE181492 | GEO

Microlunatus phosphovorus NM-1

Project description:Microlunatus phosphovorus NM-1 genome sequencing

| PRJNA66879 | ENA

MLL RIP-seq

Project description:MLL RIP-seq

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Knockdown of ILF2/ILF3 (NF45/NF90) in the gastric cancer cell line SGC7901

Project description:Our data showed that lncRNA ELF3-AS1 could bind on the exon1 of ELF3-201 to form a double-stranded RNA molecule. This double-stranded RNA could interact with ILF2/ILF3 complex. To explore the biofunction of the interaction between ELF3-AS1 and ILF2/ILF3 complex, loss-of-function studies regarding ILF2 and ILF3 were performed in SGC7901 cell line. RNA sequencing studies showed that knockdown of ILF3 significantly decreased ELF3-AS1, while knockdown of ILF2 significantly increased ELF3-AS1 and NF90 expression. Our data revealed that ILF2/ILF3 complex interacted with ELF3-AS1/ELF3 double-stranded RNA and regulated their transcripts stability.

2021-10-01 | GSE161544 | GEO

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