Epstein Barr Virus Nuclear Antigen 3C binds to BATF/IRF4/SPI1 sites and represses CDKN2A by recruiting Sin3A

ABSTRACT: We report the application of ChIP Seq to study the Epstein Barr Virus Nuclear Antigen 3C, an essential transcriptional regulator involved in the transformation of Resting B Lymphocytes to the immortalized Lymphoblast Cell Lines

ORGANISM(S): Homo sapiens

PROVIDER: GSE52632 | GEO | 2013/12/22

SECONDARY ACCESSION(S): PRJNA229507

REPOSITORIES: GEO