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Chronic stress-induced cholesterol metabolism abnormalities promote ESCC tumorigenesis and predict neoadjuvant therapy response

ABSTRACT: chronic stress activates the GCR-HMGB2-LDLR axis via cortisol release, leading to abnormal cholesterol metabolism and ultimately promoting esophageal carcinogenesis

ORGANISM(S): Homo Sapiens

SUBMITTER: Zigang Dong

PROVIDER: PXD059578 | iProX | Thu Jan 09 00:00:00 GMT 2025

REPOSITORIES: iProX

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