New mechanistic understanding of FXR agonist Vonafexor: inducing sublethal damage of HBV-positive liver cancer cells via promoting anti-tumor immunity
Ontology highlight
ABSTRACT: To elucidate the mechanism by which Vonafexor enhanced T and NK cell function, we conducted a proteomic analysis of immune cells derived from HBV-positive HCC following stimulation with Vonafexor.
ORGANISM(S): Homo Sapiens
SUBMITTER:
Banglun Pan
PROVIDER: PXD062758 | iProX | Tue Apr 08 00:00:00 BST 2025
REPOSITORIES: iProX
ACCESS DATA