Microglial NLRC5 Drives Lysosomal Dysfunction to Disrupt Autophagic Flux and Promote Post-Stroke Neuroinflammation
Ontology highlight
ABSTRACT: To further elucidate the molecular mechanisms by which microglial NLRC5 exacerbates post-stroke inflammation, we performed proteomic profiling of primary microglia isolated from WT and Nlrc5 KO mice following OGD/R treatment.
ORGANISM(S): Mus Musculus
SUBMITTER:
Xiang Cao
PROVIDER: PXD068991 | iProX | Tue Sep 30 00:00:00 BST 2025
REPOSITORIES: iProX
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