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ABSTRACT: Papillibacter cinnamivorans DSM 12816

PROVIDER: PRJEB20323 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	FWXW01.dat.gz	Other
	FWXW01.fasta.gz	Fasta.gz
	FWXW01.master.dat	Other
	FWXW01.dat.gz	Other

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Similar Datasets

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [liver]

Project description:Auricularia auricula is a well-known traditional edible and medicinal fungus with high nutritional and pharmacological values, as well as metabolic and immunoregulatory properties. However, the exact mechanisms underlying the effects of Auricularia auricula polysaccharides (AAP) on obesity and related metabolic endpoints, including the role of the gut microbiota, remain insufficiently understood. To determine the mechanistic role of the gut microbiota in observed anti-obesogenic effects AAP, faecal microbiota transplantation (FMT) and pseudo germ-free mice model treated with antibiotics were also applied, together with 16S rRNA genomic-derived taxonomic profiling. HFD murine exposure to AAP thwarted weight-gains, reduced fat depositing, together with upregulating thermogenesis proteomic biomarkers within adipose tissue. These effects were associated with diminished intestine/bloodstream-borne lipid transportation, together with enhanced glucose tolerance. FMT administered in tandem with antibiotic treatment demonstrated the intestinal microbiota was necessary in deploying AAP anti-obesogenic functions. Intestine-dwelling microbial population assessments discovered AAP to enhance (in a selective manner) Papillibacter cinnamivorans, a commensal bacterium having reduced presence within HFD mice. Notably, HFD mice treated with oral formulations of Papillibacter cinnamivorans diminished obesity and was linked to decreased intestinal lipid transportation. Datasets from the present study show that AAP thwarted dietary-driven obesity and metabolism-based disorders through regulating intestinal lipid transportation, a mechanism that is dependent on the gut commensal Papillibacter cinnamivorans. These results indicated AAP and Papillibacter cinnamivorans as newly identified pre- and probiotics that could possibly serve as novel countermeasure against obesity.

2022-05-03 | GSE201950 | GEO

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [colon]

2022-05-03 | GSE201951 | GEO

Papillibacter cinnamivorans DSM 12816

Project description:Papillibacter cinnamivorans DSM 12816 Genome sequencing

| PRJNA245693 | ENA

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans

Project description:Auricularia auricula is a well-known traditional edible and medicial fungus with high nutritional and pharmacological values, as well as metabolic and immunoregulatory properties. However, the exact mechanisms underlying the effects of Auricularia auricula polysaccharides (AAP) on obesity and related metabolic endpoints, including the role of the gut microbiota, remain insufficiently understood. To determine the mechanistic role of the gut microbiota in observed anti-obesogenic effects AAP, faecal microbiota transplantation (FMT) and pseudo germ-free mice model treated with antibiotics were also applied, together with 16S rRNA genomic-derived taxonomic profiling. HFD murine exposure to AAP thwarted weight-gains, reduced fat depositing, together with upregulating thermogenesis proteomic biomarkers within adipose tissue. These effects were associated with diminished intestine/bloodstream-borne lipid transportation, together with enhanced glucose tolerance. FMT administered in tandem with antibiotic treatment demonstrated the intestinal microbiota was necessary in deploying AAP anti-obesogenic functions. Intestine-dwelling microbial population assessments discovered AAP to enhance (in a selective manner) Papillibacter cinnamivorans, a commensal bacterium having reduced presence within HFD mice. Notably, HFD mice treated with oral formulations of Papillibacter cinnamivorans diminished obesity and was linked to decreased intestinal lipid transportation. Datasets from the present study show that AAP thwarted dietary-driven obesity and metabolism-based disorders through regulating intestinal lipid transportation, a mechanism that is dependent on the gut commensal Papillibacter cinnamivorans. These results indicated AAP and Papillibacter cinnamivorans as newly identified pre- and probiotics that could possibly serve as novel countermeasure against obesity

2022-09-16 | MTBLS4469 | MetaboLights

Project description:Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans

| PRJNA814477 | ENA

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans

Project description:Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans

| PRJNA833725 | ENA

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [colon]

Project description:Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [colon]

| PRJNA839171 | ENA

Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [liver]

Project description:Auricularia auricula polysaccharides reduce obesity in mice through gut commensal Papillibacter cinnamivorans [liver]

| PRJNA839266 | ENA

Expression analysis of strains Lactococcus lactis KCTC 3769T, L. raffinolactis DSM 20443T, L. plantarum DSM 20686T, L. fujiensis JSM 16395T, L. garvieae KCTC 3772T, L. piscium DSM 6634T and L. chungangensis CAU 28T

2014-05-28 | GSE58002 | GEO

Project description:Investigation of whole genome gene expression level changes in Lactococcus lactis KCTC 3769T,L. raffinolactis DSM 20443T, L. plantarum DSM 20686T, L. fujiensis JSM 16395T, L. garvieae KCTC 3772T, L. piscium DSM 6634T and L. chungangensis CAU 28T . This proves that transcriptional profiling can facilitate in elucidating the genetic distance between closely related strains. A one chip study using total RNA recovered from of L. raffinolactis DSM 20443T, L. plantarum DSM 20686T, L. fujiensis JSM 16395T, L. garvieae KCTC 3772T, L. piscium DSM 6634T and L. chungangensis CAU 28T . For the the transcriptome of of L. raffinolactis DSM 20443T, L. plantarum DSM 20686T, L. fujiensis JSM 16395T, L. garvieae KCTC 3772T, L. piscium DSM 6634T and L. chungangensis CAU 28T was analyzed using the Lactococcus lactis KCTC 3769T microarray platform

2014-05-28 | E-GEOD-58002 | biostudies-arrayexpress

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