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Expression profile of RCC4 cells with YAP silencing

ABSTRACT: Expression profile of RCC4 cells with YAP silencing

PROVIDER: PRJNA610236 | ENA |

REPOSITORIES: ENA

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Expression profile of RCC4 cells with YAP silencing

Project description:YAP is the one of the effectors of Hippo pathway. YAP promotes organ size growth as well as tumor metastasis. In this study, YAP is silencing in clear cell carcinoma cell line RCC4 to elucidate the downstream targets that promotes the resisntance toward erastin-induced ferroptosis.

2020-03-05 | GSE146354 | GEO

Expression profile of MESH1 and ATF4 silencing RCC4 cells

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Expression profile of RCC4 cells with TAZ (WWTR1) silencing

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Expression profile of RCC4 cells with TAZ (WWTR1) silencing

Project description:TAZ, also known as WWTR1, is the one of the effectors of Hippo pathway. With its paralog, YAP, TAZ promotes organ size growth as well as tumor metastasis. In human renal carcinoma cells, we found that TAZ-silencing induces resisntance toward erastin-induced ferroptosis. In this study, TAZ is silencing in clear cell carcinoma cell line RCC4 to elucidate the downstream targets that promotes the resistance toward erastin-induced ferroptosis.

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Transcriptional profile of CAOV2 ovarian cancer cells with YAP silencing

Project description:Transcriptional profile of CAOV2 ovarian cancer cells with YAP silencing

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Transcriptional profile of CAOV2 ovarian cancer cells with YAP silencing

Project description:We performed RNAseq to study the transcriptional changes of human CAOV2 ovarian cancer cells with or without YAP silencing by siRNAs to understand the genetic determinants of ferroptosis response

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Project description:MESH1 encodes for Metazoan SpoT Homolog 1, which is a homologue of bacterial SpoT that mediates bacterial stringent response. In human cells, we found that MESH1-silencing induces an extensive transcriptional response in clear cell carcinoma cell line RCC4 that partially overlap with mammalian cell integrated stress response (ISR), which ATF4 up-regulation is an essential branch of the response. In this study, the goal is to elucidate the role of ATF4 up-regulation in MESH1-silencing transcriptional response.

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Transcriptional profile of MESH1-silenced RCC4 cells

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Transcriptional profile of recurrent breast cancer cells with RIPK3 silencing

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