Pathophysiology of arterial cardiovascular events in patients with JAK2V617F myeloproliferative neoplasms
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ABSTRACT: Cardiovascular events are the leading cause of death in patients with JAK2V617F myeloproliferative neoplasms. Their mechanisms are poorly understood. To investigate the role of microvesicles in these events, we performed a proteomic analysis of microvesicles derived from red blood cells from mice with a myeloproliferative neoplasms (Jak2V617F Flex/WT ;VE-cadherin-Cre) vs. littermate controls.
INSTRUMENT(S):
ORGANISM(S): Mus Musculus (mouse)
TISSUE(S): Blood Plasma, Extracellular Vesicle
SUBMITTER:
Valentin SABATET
LAB HEAD: Damarys Loew
PROVIDER: PXD014451 | Pride | 2020-02-17
REPOSITORIES: Pride
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