Pull down of full length and mutant BCL9 (deltaHD1) in mouse colorectal tumors and colonic epithelium
Ontology highlight
ABSTRACT: Bcl9 and Bcl9l (Bcl9l) are commonly thought as beta-catenin-dependent Wnt signaling transcriptional activators. Here we searched for BCL9 interacts that could explain the PYGO-independent phenotype induced be the loss of the HD1 (PYGO-binding) domain of BCL9
INSTRUMENT(S): Q Exactive HF
ORGANISM(S): Mus Musculus (mouse)
TISSUE(S): Epithelial Cell, Colon
DISEASE(S): Colon Cancer
SUBMITTER: Claudio Cantù
LAB HEAD: Claudio Cantù
PROVIDER: PXD018805 | Pride | 2020-08-10
REPOSITORIES: Pride
ACCESS DATA