In planta expression of human polyQ-expanded huntingtin fragment reveals mechanisms to prevent disease-related protein aggregation.
Ontology highlight
ABSTRACT: We have Q67 worms with overexpression of SPP construct or without overexpression of SPP construct. We compare whole proteome from control group with worms overexpressing of SPP construct
INSTRUMENT(S):
ORGANISM(S): Caenorhabditis Elegans
DISEASE(S): Huntington Disease
SUBMITTER:
Proteomics Facility
LAB HEAD: David Vilchez
PROVIDER: PXD044145 | Pride | 2023-08-11
REPOSITORIES: Pride
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