Ontology highlight
ABSTRACT:
INSTRUMENT(S):
ORGANISM(S): Homo Sapiens (human)
TISSUE(S): Blood Serum
DISEASE(S): Iga Glomerulonephritis
SUBMITTER:
Niyaz Alsharabi
LAB HEAD: Øystein Eikrem
PROVIDER: PXD065216 | Pride | 2026-02-09
REPOSITORIES: Pride
| Action | DRS | |||
|---|---|---|---|---|
| Jessica_F_IgG_Ex1_F.mzML | Mzml | |||
| Jessica_F_IgG_Ex1_F.mzid | Mzid | |||
| Jessica_F_IgG_Ex1_F1.raw | Raw | |||
| Jessica_F_IgG_Ex1_F10.raw | Raw | |||
| Jessica_F_IgG_Ex1_F2.raw | Raw |
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Chen Tony Jialiang TJ Skandalou Eleni E Fritz-Wallace Katarina K Apeland Terje T Knoop Thomas T Marti Hans-Peter HP Eikrem Øystein Ø Furriol Jessica J
Clinical kidney journal 20251111 12
<h4>Background</h4>Immunoglobulin A nephropathy (IgAN) is a common cause of chronic kidney disease (CKD) and identifying early molecular signatures is crucial for IgAN risk stratification and timely intervention. In this study we used serum proteomics to investigate molecular differences between IgAN patients who progressed and those who remained stable before separation in kidney function became apparent.<h4>Methods</h4>Serum samples from 40 adults with biopsy-proven IgAN were analysed and clas ...[more]