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A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [RNA-seq]

ABSTRACT: A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [RNA-seq]

PROVIDER: PRJNA1132521 | ENA |

REPOSITORIES: ENA

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A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception

Project description:A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception

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A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [ChIP-seq]

Project description:A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [ChIP-seq]

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A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [ChIP-seq]

Project description:OsWRKY53 responded to both wounding and wound-derived peptide, OsPep2 treatment. We have performed ChIP-seq experiments to determine the contribution of OsWRKY53 to both wounding as well as OsPep2 treatment.

2025-09-02 | GSE271624 | GEO

A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception

Project description:This SuperSeries is composed of the SubSeries listed below.

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A wound-responsive transcription factor, OsWRKY53 dictates downstream wound responses upon OsPep2 perception [RNA-seq]

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Evaporative cooling signals for wound healing in plants

Project description:Repairing a damaged body part is critical for the survival of an organism. Tissue damage induces rapid responses to activate downstream events including defense, regeneration and wound healing. Despite accumulating knowledge of early wound signaling including the orchestrated actions of phytohormones, electric circus and reactive oxygen species, our knowledge about the end point of a wound response - wound healing, is still limited. We observed that a local temperature reduction associated with the activation of cold-responsive genes occurred at wounding site on Arabidopsis leaves, which was likely caused by evaporative cooling. The disappearance of localized cooling and restoration of cold responsive genes to a steady state could be used as a quantitative readout of wound healing. Based on these observations, we developed a deep learning pipeline to monitor the dynamics of wound healing. We found that CBFs transcription factors relay injury-induced cooling signal to wound healing. Thus, our work provides a tool to quantify wound healing in plants and advances our understanding of tissue repair in plants.

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