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Pseudomonas syringae pv. actinidiae strain:Psa NZ-47

ABSTRACT: Pseudomonas syringae pv. actinidiae strain:Psa NZ-47 Genome sequencing and assembly

PROVIDER: PRJNA339322 | ENA |

REPOSITORIES: ENA

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			Action	DRS
		Other
	SRR5010915_subreads.fastq.gz	Fastqsanger.gz
	SRR5010916_subreads.fastq.gz	Fastqsanger.gz
	SRR5010917_1.fastq.gz	Fastqsanger.gz
	SRR5010917_2.fastq.gz	Fastqsanger.gz

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Pseudomonas syringae pv. actinidiae strain:Psa NZ-63

Project description:Pseudomonas syringae pv. actinidiae NZ-63 ICMP 22183

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Whole-transcriptome microarray analysis of Pseudomonas syringae pv. actinidiae (Psa) treated with Gunpowder green tea extract (EGCG 0.4 mg/ml)

Project description:Purpose: Microarray technologies provide a unique opportunity to deeply investigate bacterial molecular responses to treatments. Pseudomonas syringae pv. actinidiae (Psa) is the causal agent of the bacterial canker of kiwifruit causing severe economic losses worldwide. At present, integrated control strategies include chemical treatments with copper-based products and preventive measures but the high virulence and fast spreading of the bacterium are hardly controlled by such measures, and especially copper use is questioned because of the possible appearance of copper resistant bacterial strains. The present project aims at the identification of Psa responses to green tea treatment (Gunpowder variety) at sub-lethal concentration (0.4 mg/ml). Methods: Psa cells were cultured in liquid KB (controls) or in KB supplemented with Gunpowder tea (Gunpowder-trateted) at 0.4 mg/ml EGCG for 24 h at 28°C. The microarray experiments on Gunpowder treated or untreated samples in biological triplicate resulted in 6 samples to be analyzed. Conclusions: This work identified important molecular mechanisms involved in Psa responses upon Gunpowder green tea treatment.

2019-09-23 | GSE137806 | GEO

Pseudomonas syringae pv. actinidiae strain:Psa NZ-66

Project description:Pseudomonas syringae pv. actinidiae Genome sequencie and assembly

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Pseudomonas syringae pv. actinidiae strain:Psa 105743

Project description:Pseudomonas syringae pv. actinidiae strain:Psa 105743 Genome sequencing and assembly

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Pseudomonas syringae pv. actinidiae strain:Psa 127771

Project description:Pseudomonas syringae pv. actinidiae strain:Psa 127771 Genome sequencing and assembly

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Pseudomonas syringae pv. actinidiae strain:Psa NZ-64

Project description:Pseudomonas syringae pv. actinidiae ICMP 22183 Genome sequence and assembly

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Pseudomonas syringae pv. actinidiae strain:Psa NZ-65

Project description:Pseudomonas syringae pv. actinidiae ICMP 22184 Genome sequence and assembly

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Pseudomonas syringae pv. actinidiae strain:Psa NZ-62

Project description:Pseudomonas syringae pv. actinidiae ICMP 22181 Genome sequence and assembly

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Whole-transcriptome microarray analysis of different biovars of Pseudomonas syringae pv. actinidiae grown in rich medium or apoplast-like conditions

Project description:Purpose: Pseudomonas syringae pv. actinidiae (Psa) is a phytopathogen that causes devastating bacterial canker in kiwifruit. Among five biovars defined by genetic, biochemical and virulence traits, Psa3 is the most aggressive and is responsible for the most recent reported outbreaks, but the molecular basis of its heightened virulence is unclear. A custom P. syringae multi-strain whole-genome microarray platform, encompassing biovars Psa1, Psa2 and Psa3 and the well-established model P. syringae pv. tomato, was used to analyse early bacterial responses to an apoplast-like minimal medium. Conlusion: this work highlighted that diverse early responses to the host apoplast, even among bacteria belonging to the same pathovar, can lead to different virulence strategies and may explain the differing outcomes of infections.

2021-01-09 | GSE164472 | GEO

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