Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Pseudoglutamicibacter cumminsii strain:IME1328

ABSTRACT: Draft Genome Sequence of Arthrobacter cumminsii IME1328, Isolated from a Patient who Suffered from Foot Ulcers in China

PROVIDER: PRJNA385709 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
		Other

Items per page:

1 - 1 of 1

Similar Datasets

Pseudoglutamicibacter cumminsii

Project description:Arthrobacter cumminsii overview

| PRJNA184617 | ENA

Pseudoglutamicibacter cumminsii strain:XJ001

Project description:Pseudoglutamicibacter cumminsii strain:XJ001 Genome sequencing and assembly

| PRJNA1044933 | ENA

Arthrobacter cumminsii ws_2216

Project description:Arthrobacter cumminsii ws_2216 genome sequence

| PRJEB361 | ENA

Unusual isolation of Pseudoglutamicibacter cumminsii in urine culture.

Project description: Not available

| S-EPMC8790646 | biostudies-literature

Pseudoglutamicibacter cumminsii strain:DSM 10493 | isolate:DSM 10493

Project description:Pseudoglutamicibacter cumminsii DSM 10493 genome sequencing

| PRJNA695667 | ENA

Isolation and whole genomic analysis of mesophilic bacterium <i>Pseudoglutamicibacter cumminsii</i> in epithelial mesothelioma.

Project description: Not available

| S-EPMC11336841 | biostudies-literature

Circular RNA expression profiling in diabetic foot ulcers and human normal acute wounds

Project description:Circular RNA (circRNA) microarray analysis was performed to examine the expression profiles of circRNAs in diabetic foot ulcers (DFU) and in human excisional skin wounds 7 days after injury.

2019-10-01 | GSE114248 | GEO

Overexpression of hsa-HLA-DRB1 may play a role in diabetic foot ulcers by regulating miRNA_12118 and FLT-1

Project description:Introduction: We investigated the role and mechanism of hsa-HLA-DRB1 in the development and progression of diabetic foot ulcers. Methods: High-throughput sequencing was performed on three normal foot trauma tissues and diabetic foot ulcer tissues. The circRNAs with significant differences were identified. The downstream miRNAs were predicted by miRanda and RNAhybrid databases, and the mRNAs were predicted by the TargetScan database. Validation was performed with CCK8, flow cytometry, trabecular scratch assay, tubule generation assay, Western blot, dual luciferase assay, and RT-qPCR. Results: High-throughput sequencing identified 461 significantly different circRNAs, of which 260 were up-regulated and 201 down-regulated. Compared to normal tissue, hsa-HLA-DRB1 was highly expressed in diabetic foot ulcers. The hsa-HLA-DRB1/miRNA_12118/FLT-1 axis was constructed. In vitro, we found that HLA-DRB1 overexpression inhibited cell viability, wound healing, and tubule formation, promoted apoptosis, and enhanced FLT-1 expression in HUVECs. Conclusion: The upregulation of hsa-HLA-DRB1 may promote diabetic foot development by targeting miRNA_12118 and acting on FLT-1. Therefore, our study highlights the key role of the hsa-HLA-DRB1/miRNA_12118/FLT-1 axis in diabetic foot trauma.

2025-06-04 | GSE286165 | GEO

Project description:Skin microbiota of diabetic foot ulcers

| PRJNA1451799 | ENA

Project description:WGS of S. aureus from Diabetic Foot Ulcers

| PRJNA1442169 | ENA