Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Differential Gene Expression in Cdh13 KO x Maternally separated mice

ABSTRACT: Differential Gene Expression in Cdh13 KO x Maternally separated mice

PROVIDER: PRJNA488069 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR7758550.fastq.gz	Fastqsanger.gz
	SRR7758560.fastq.gz	Fastqsanger.gz
	SRR7758570.fastq.gz	Fastqsanger.gz
	SRR7758580.fastq.gz	Fastqsanger.gz
	SRR7758590.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 69

Similar Datasets

Differential Gene Expression in Cdh13 KO x Maternally separated mice

Project description:We report the data of a GxE experiment. Cdh13 ko vs Cdh13 wt, who were either maternally separated or controls who were handled. Two age groups were created but analysed separatly.

2021-01-30 | GSE119082 | GEO

Differential Gene Expression in Adgrl3 KO Mice

Project description:Differential Gene Expression in Adgrl3 KO Mice

| PRJNA481875 | ENA

Differential expression of GIF KO vs WT mice

Project description:Differential expression of GIF KO vs WT mice in Liver and Bone with special scope in IGF signaling pathway. This data is part of a pre-publication release. For information on the proper use of pre-publication data shared by the Wellcome Trust Sanger Institute (including details of any publication moratoria), please see http://www.sanger.ac.uk/datasharing/

2013-06-11 | E-ERAD-163 | biostudies-arrayexpress

Differential gene expression in Gaddβ KO mice treated with diethylnitrosamine and phenobarbital

Project description:Differential gene expression in Gaddβ KO mice treated with diethylnitrosamine and phenobarbital

| PRJNA967695 | ENA

The differential gene expression of the dentate gyrus between WT and KO mice as well as between Fst knockout and overexpressing mice

Project description:we aim to find the possible pathway and mechanism whereby Fst mediates neurogenesis and spatial learning by utilising RNA-seq to identify differential hippocampal gene expression between WT and KO mice as well as between KO and Fst overexpression mice

2020-12-18 | GSE163449 | GEO

Transcriptome profiling of kidney medulla of Actr3 KO mice

Project description:Renal tubule-specific Actr3 knockout mice (Actr3-fl/fl*Cdh16-Cre) were generated. Transcriptome profiling (RNA-Seq) and differential gene expression analysis of isolated renal medulla from KO and WT mice at p14 was performed. Actr3 KO mice showed transcriptional changes associated with Actr3 loss and kidney disease.

2025-10-16 | GSE287609 | GEO

Lysosome-enriched fractions from the liver of Cln8 KO mice and WT mice

Project description:Lysosome-enriched fractions from the liver of Cln8 KO mice and WT mice. Included are four datasets: 1. Lysosome-enriched fraction from the liver of Cln8 KO mice, replicate 1 (CLN8_KO_1). 2. Lysosome-enriched fraction from the liver of Cln8 KO mice, replicate 2 (CLN8_KO_2). 3. Lysosome-enriched fraction from the liver of WT mice, replicate 1 (WT_1). 4. Lysosome-enriched fraction from the liver of WT mice, replicate 2 (WT_2).

| MSV000082938 | MassIVE

Differential Gene Expression in Adgrl3 KO Mice

Project description:We report the differential expression of genes in Adgrl3 -/- mice in comparison to Adgrl3 +/+ mice. Three ADHD-relevant brain regions; hippocampus, pre-frontal cortex and striatum are included alongside a whole-brain analysis

2021-01-30 | GSE117357 | GEO

Maternally inherited piRNAs silence transposons during Drosophila embryogenesis

Project description:The piRNA pathway controls transposon expression in animal germ cells, thereby ensuring genome stability over generations. piRNAs are maternally deposited and required for proper transposon silencing in adult offspring. However, a long-standing question in the field is the precise function of maternally deposited piRNAs and its associated factors during embryogenesis. Here, we probe the spatio-temporal expression patterns of several piRNA pathway components during early stages of development. Amongst those, factors required for transcriptional gene silencing (TGS) showed ubiquitous abundance in somatic and pole cells throughout the first half of embryogenesis. We further analysed the transcriptomes of various embryo stages and correlated these with the presence of selected chromatin marks. We found that a number of transposon families show bursts of transcription during early embryonic stages. Transposons heavily targeted by maternally deposited piRNAs accumulated repressive chromatin marks following their spike in expression. Furthermore, depletion of maternally deposited Piwi protein in early embryos resulted in increased expression of transposons targeted by inherited piRNAs and was accompanied by a strong loss of repressive chromatin marks at coding sequences. Overall, our data suggests a pivotal role for the piRNA pathway in transposon defence during Drosophila embryogenesis in somatic cells.

2021-07-26 | PXD022409 | Pride

Differential gene expression in the airway epithelium of beta6 ko mice chronically challenged with OVA

Project description:Expression microarrays identified 119 genes that were significantly differentially expressed in the epithelium of WT and M-NM-26 KO mice after saline or chronic allergen challenge. PAM clustering revealed two interesting clusters (6 and 8) that we had not identified in previous comparisons by whole lung microarrays. Cluster 6 genes were low at baseline in both WT and M-NM-26 KO mice and were increased in WT but not in M-NM-26 KO mice after chronic allergen challenge such as Mcpt1. Cluster 8 genes were increased at baseline in M-NM-26 KO mice, and included 6 mast cell related genes (cma1, mcpt4, cpa3, mcpt6, tpsab1, il1rl1). The most informative differentially expressed genes identified in microarrays of the epithelial microenvironment were not epithelial genes, but mast cell genes. Control and beta6 ko mice (wt-S (4), wt-OVA (4), ko-S (4), ko-OVA (4)) were sensitized and challenged with OVA or saline, airway epithelium were brush-harvested.

2012-12-31 | E-GEOD-28168 | biostudies-arrayexpress

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data