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A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab [ribosome profiling]

ABSTRACT: A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab [ribosome profiling]

PROVIDER: PRJNA527204 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR8732200.fastq.gz	Fastqsanger.gz
	SRR8732210.fastq.gz	Fastqsanger.gz
	SRR8732201.fastq.gz	Fastqsanger.gz
	SRR8732211.fastq.gz	Fastqsanger.gz
	SRR8732202.fastq.gz	Fastqsanger.gz

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A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab

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A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab

Project description:This SuperSeries is composed of the SubSeries listed below.

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A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab [ribosome profiling]

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A large panel of isogenic APP and PSEN1 mutant human iPSC neurons reveals shared endosomal abnormalities mediated by APP b-CTFs, not Ab [RNA-seq]

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