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Transcriptomic profile of the FERONIA mutant

ABSTRACT: Transcriptomic profile of the FERONIA mutant

PROVIDER: PRJNA601158 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR10883941.fastq.gz	Fastqsanger.gz
	SRR10883942.fastq.gz	Fastqsanger.gz
	SRR10883943.fastq.gz	Fastqsanger.gz
	SRR10883944.fastq.gz	Fastqsanger.gz
	SRR10883945.fastq.gz	Fastqsanger.gz

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Project description:FERONIA (FER) receptor kinase has a versatile role in plant growth and development, biotic and abiotic stress responses, and reproduction. To gain new insights into the molecular interplay of these processes and to identify new functions of FER, we carried out quantitative transcriptome, proteome, and phosphoproteome profiling of wild type (WT) and a loss-of-function fer mutant in Arabidopsis plants. Gene Ontology terms for hormone signaling, abiotic stress, and biotic stress were significantly enriched among mis-expressed transcripts, proteins, and/or mis-phosphorylated proteins, in agreement with FER’s known roles in these processes. Analysis of multi-omics data and subsequent experimental evidence identified previously unknown functions of FER in ER (Endoplasmic Reticulum) body formation, glucosinolate biosynthesis. FER functions through transcription factor NAI1 to mediate ER body. FER also negatively regulates indole glucosinolates biosynthesis, partially through NAI1. Furthermore, we found that a group of abscisic acid (ABA)-induced transcription factors are hypo-phosphorylated in the fer mutant and demonstrated that FER acts through ABI5 to negatively regulate the ABA response during germination. Our integrated-omics study therefore reveals novel functions of FER and provides new insights into the underlying mechanisms of FER function.

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Project description:High humidity greatly influences plant growth and development by triggering adaptive physiological responses such as leaf hyponasty. Recent studies identified CNGC2/4-mediated Ca2+ influx and CAMTA2/3-mediated transcription as essential for this process, but the upstream regulators that control these pathways remain unknown. Here, we show that the receptor-like kinase FERONIA (FER) is required for Arabidopsis adaptation to high humidity. Transcriptomic profiling revealed rapid induction of CNGC2, CAMTA targets, and pectin remodeling genes in wild-type plants but not in fer-4 loss of function mutants. A targeted mutant screen confirmed that FER and its co-receptor LORELEI-LIKE GPI-ANCHORED PROTEIN1 (LLG1) are essential for humidity-induced hyponasty. Whole-plant Ca²⁺ imaging demonstrated that high humidity triggers conserved cytosolic Ca²⁺ elevations across species, including petiole-localized Ca²⁺ waves in Arabidopsis that precede hyponastic movement by ~2 h. These signals were significantly attenuated in fer-4, providing a mechanistic explanation for its transcriptional and physiological defects. Together, our findings identify FERONIA as a key upstream component of a sensing mechanism linking extracellular high humidity to Ca²⁺ signaling, cell wall remodeling, and adaptive leaf movement.

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Project description:The goal of this study was to determine protein abundance and phosphorylation changes in the FERONIA mutant (fer) in Arabidopsis leaves. Seeds were germinated on 1/2MS with constant light (24hr light) for 10 days. Then, plants were transferred to soil and grown under short day condition (8-hour light and 16-hour dark) at 22C for another 3 weeks (21 days). Samples were 4 weeks old when collected. Three biological replicates of WT as well as fer mutant rosette leaves were collected at 4 weeks. Proteins were extracted and processed into peptides using a phenol-FASP methods (https://doi.org/10.1002/pmic.201800220). Peptides were labeled with TMT6-plex reagents (WT 126, 127N, 128C; fer 129N, 130C, 131). Following TMT multiplexing phoshopeptides were enriched using Titanspere Phos-TiO 10 uM beads (GL science).

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