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GenomicsA novel mRNA decay inhibitor abolish pathophysiological cellular transition
A novel mRNA decay inhibitor abolish pathophysiological cellular transition
PRJNA805270 | ENA
UnknownMutation of a conserved glutamine residue does not abolish desensitization of acid-sensing ion channel 1.
Not available
S-EPMC8167889 | biostudies-literature
GenomicsALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function
ALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function
PRJNA805280 | ENA
UnknownSulfonamide-Based Inhibitors of Aminoglycoside Acetyltransferase Eis Abolish Resistance to Kanamycin in Mycobacterium tuberculosis.
Not available
S-EPMC5154685 | biostudies-literature
UnknownIdentification and characterization of MUS81 point mutations that abolish interaction with the SLX4 scaffold protein.
Not available
S-EPMC4251979 | biostudies-literature
GenomicsALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function [RNA-seq]
ALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function [RNA-seq]
PRJNA805277 | ENA
GenomicsALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function [RIP-seq]
ALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function [RIP-seq]
PRJNA805281 | ENA
UnknownBML-265 and Tyrphostin AG1478 Disperse the Golgi Apparatus and Abolish Protein Transport in Human Cells.
Not available
S-EPMC6797785 | biostudies-literature
TranscriptomicsAn alternative, arginase-independent pathway for arginine metabolism in Kluyveromyces lactis involves guanidinobutyrase as a key enzyme
Most available knowledge on fungal arginine metabolism is derived from studies on Saccharomyces cerevisiae, in which arginine catabolism is initiated by releasing urea via the arginase reaction. Orthologs of the S. cerevisiae genes encoding the first three enzymes in the arginase pathway were cloned...
ORGANISM(S): Kluyveromyces lactis 
2014-07-16 | E-GEOD-56060 | biostudies-arrayexpress
ProteomicsALS-Associated KIF5A Mutations Abolish Autoinhibition Resulting in a Toxic Gain of Function
Understanding the pathogenic mechanisms of disease mutations is critical to advancing treatments. ALS-associated mutations in the gene encoding the microtubule motor KIF5A result in skipping of exon 27 (KIF5AΔExon27) and the encoding of a protein with a novel 39-amino acid residue C-terminal sequenc...
ORGANISM(S): Homo sapiens (Human) 
2022-06-09 | PXD031012 | Pride
Human Kif5a Interactors Als 
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