myh7 - OmicsDI

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Faculty of Technical Chemistry, Institute of Chemical Technologies and Analytics, Technische Universität Wien (TU Wien), 1060 Vienna, Austria; Diagnostic and Research Institute of Pathology, Medical University of Graz, 8010 Graz, Austria; BiotechMed-Graz, 8010 Graz, Austria; (1)
Institute for Clinical Chemistry and Pathobiochemistry, Department for Diagnostic Laboratory Medicine, University Hospital Tübingen, Tübingen, Germany (1)
Prof. Nir Eynon, Australian Regenerative Medicine Institute, Monash University, Clayton VIC 3800, Australia.Proteomics contact: Dr Joel R. Steele, Monash Proteomics & Metabolomics Platform, Monash Biomedicine Discovery Institute, Monash University, Clayton VIC, Australia (1)
Venetian Institute of Molecular Medicine (VIMM), Via Orus 2, 35129, Padova, Italy Department of Biomedical Sciences, 35137, University of Padova, Padova, Italy (1)

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MYH7 G256E hiPSC-CM (Day 30)

PRJNA912105 | ENA

Beta-Adrenergic Stimulation and MYH7 G256E Mutant Gene Dosage Drive Hypertrophic Cardiomyopathy Phenotype Penetrance

PRJNA1155708 | ENA

MYH7 G256E hiPSC-CM (Day 30)

ORGANISM(S): Homo sapiens

2024-04-15 | GSE220933 | GEO

Genome sequencing and variant calling of HCM Patients with MYH7 variants

PRJNA1251724 | ENA

Genome sequencing and variant calling of HCM Patients with MYH7 variants

PRJNA1251723 | ENA

Beta-Adrenergic Stimulation and MYH7 G256E Mutant Gene Dosage Drive Hypertrophic Cardiomyopathy Phenotype Penetrance

We characterized the baseline transcriptomic phenotype of day30 iPSC-Cardiomyocytes carrying hypertrophic cardiomyopathy associated heterozygous mutations MYH7 G256E and H251N using scRNAseq. We additionally analzyed trancriptomic signatures of HOM MYH7 G256E iPSC-CMs, and of HET MYH7 G256E iPSC-CMs...

ORGANISM(S): Homo sapiens

2025-08-01 | GSE276210 | GEO

MYH7 Mutations in Restrictive Cardiomyopathy.

Not available

S-EPMC12102527 | biostudies-literature

A long noncoding RNA protects the heart from pathological hypertrophy

The role of long noncoding RNA (lncRNA) in adult hearts is unknown; also unclear is how lncRNA modulates nucleosome remodelling. An estimated 70% of mouse genes undergo antisense transcription1, including myosin heavy chain 7 (Myh7), which encodes molecular motor proteins for heart contraction2. Her...

ORGANISM(S): Homo sapiens

2014-08-05 | E-GEOD-49716 | biostudies-arrayexpress

Allele-Selective Knockdown of MYH7 Using Antisense Oligonucleotides.

Not available

S-EPMC7033438 | biostudies-literature

A case report of MYH7 mutation-induced restrictive cardiomyopathy.

Not available

S-EPMC12023744 | biostudies-literature