p16 knockdown - OmicsDI

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Linking immortalization to direct conversion of human fibroblasts to neurons

Direct conversion from fibroblasts to neurons is a potential cell replacement therapy for neurological disorders, and a variety of combinations of transcription factors have been tried. We notice that the efficiency of conversion from aging fibroblasts was much lower than in early stage cells, which...

ORGANISM(S): Homo sapiens

2012-12-28 | E-GEOD-43175 | biostudies-arrayexpress

Knockdown of SR protein family in P19 cells and RNA-seq of cytoplasmic and whole cell polyA+ RNA

Our data suggest that all SR proteins contribute to mRNA export via NXF1. To identify endogenous export targets we depleted all seven SR proteins individually from P19 WT cells prepared cytoplasmic fractions. We sequenced the cytoplasmic fraction and as a control whole celll RNA from the identical s...

ORGANISM(S): Mus musculus

2016-03-07 | E-GEOD-69733 | biostudies-arrayexpress

IFI30 Knockdown Inhibits ESCC Progression by Promoting Apoptosis and Senescence via Activation of JNK and P21/P16 Pathways.

Not available

S-EPMC11971534 | biostudies-literature

Suppression of p16 alleviates the senescence-associated secretory phenotype.

Not available

S-EPMC7906185 | biostudies-literature

De novo purine metabolism is a metabolic vulnerability of cancers with low p16 expression.

Not available

S-EPMC10369956 | biostudies-literature

The tRNA methyltransferase NSun2 stabilizes p16INK⁴ mRNA by methylating the 3'-untranslated region of p16.