Sort   by:  
 Page size 
ProteomicsGlobal proteome and phosphoproteome characterization of sepsis-induced kidney injury
Sepsis-induced acute kidney injury (S-AKI) is the most common complication in hospitalized and critically ill patients, highlighted by a rapid decline of kidney function occurring a few hours or days after sepsis onset. Systemic inflammation elicited by microbial infections is believed to lead to ki...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
MSV000086126 | MassIVE
ProteomicsPlasma proteomics defines two reproducible subphenotypes of sepsis-associated acute kidney injury with distinct outcomes
This dataset contains de-identified Olink proteomics data from the ADRENOSS cohort used in the manuscript "Plasma proteomics defines two reproducible subphenotypes of sepsis-associated acute kidney injury with distinct outcomes". The dataset includes plasma protein measurements generated using the O...
ORGANISM(S): Homo sapiens (Human) 
2026-07-28 | PAD000052 | Pride
Olink  affinity proteomics  adrenoss  sepsis-associated acute kidney injury  plasma proteomics  clustering  subphenotypes  biomarkers 
ProteomicsSAA1 delivered by tubular epithelial cell-derived extracellular vesicles aggravate sepsis-associated acute kidney injury by promoting NET formation
Sepsis-associated acute kidney injury (SA-AKI) is highly lethal with a rapid onset, and effective drugs for treatment are lack due to the molecular pathogenesis underlying SA-AKI remains elusive. Tubular epithelial cells (TECs) have increasingly been recognized as a driving force in the progression ...
ORGANISM(S): Mus Musculus 
PXD055037 |
GenomicsSepsis-associated acute kidney injury in mice and humans elicits heterogeneous renal microvascular microRNA responses
Sepsis-associated acute kidney injury in mice and humans elicits heterogeneous renal microvascular microRNA responses
PRJNA934507 | ENA
GenomicsLow-level tragus stimulation alleviates sepsis-associated acute kidney injury by promoting renal M2 macrophage polarization.
Low-level tragus stimulation alleviates sepsis-associated acute kidney injury by promoting renal M2 macrophage polarization.
PRJNA1281050 | ENA
GenomicsThymosin β4 alleviates sepsis-associated acute kidney injury by suppressing MAPK signaling pathway
Thymosin β4 alleviates sepsis-associated acute kidney injury by suppressing MAPK signaling pathway
PRJNA1456092 | ENA
GenomicsThe effect of growth differentiation factor 11 on sepsis-associated acute kidney injury
The effect of growth differentiation factor 11 on sepsis-associated acute kidney injury
PRJNA1265641 | ENA
GenomicsMyeloid-specific ferritin light chain deletion does not confer renal protection in sepsis-associated acute kidney injury
Myeloid-specific ferritin light chain deletion does not confer renal protection in sepsis-associated acute kidney injury
PRJNA1074352 | ENA
ProteomicsProteomics and phosphoproteomics of sepsis induced acute and chronic kidney disease
Sepsis induced acute kidney injury (AKI) is associated with a rapid decline of kidney function, leading to unacceptably high morbidity and mortality rate as well as progression to chronic kidney disease (CKD). We have recently developed a septic AKI mouse model. By quantitatively assessing the kidne...
ORGANISM(S): Mus musculus (Mouse) 
2020-09-25 | PXD017045 | Pride
Proteomics  phosphoproteomics  sepsis  kidney  aki  ckd 
UnknownSepsis-Associated Acute Kidney Injury.
Not available
S-EPMC7995616 | biostudies-literature
Sort   by:  
 Page size