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GenomicsTranscriptome profiling of NF1 MPNST line RHT-172 after ZNF423 siRNA knockdown
Transcriptome profiling of NF1 MPNST line RHT-172 after ZNF423 siRNA knockdown
PRJNA1427191 | ENA
GenomicsTranscriptome profiling of NF1 MPNST line ST88-14 (8814) after ZNF423 shRNA knockdown
Transcriptome profiling of NF1 MPNST line ST88-14 (8814) after ZNF423 shRNA knockdown
PRJNA1427179 | ENA
GenomicsTranscriptome profiling of NF1 MPNST line NF90-8 (90.8) after ZNF423 siRNA knockdown
Transcriptome profiling of NF1 MPNST line NF90-8 (90.8) after ZNF423 siRNA knockdown
PRJNA1427190 | ENA
GenomicsTranscriptome profiling of NF1 MPNST line JH-2-002 after ZNF423 siRNA knockdown
Transcriptome profiling of NF1 MPNST line JH-2-002 after ZNF423 siRNA knockdown
PRJNA1426372 | ENA
GenomicsTranscriptome profiling of NF1 MPNST line ST88-14 (8814) after ZNF423 siRNA knockdown
Transcriptome profiling of NF1 MPNST line ST88-14 (8814) after ZNF423 siRNA knockdown
PRJNA1426415 | ENA
TranscriptomicsGene expression analysis of SH-SY5Y neuroblastoma cells overexpressing ZNF423
We have identified ZNF423 (also known as Ebfaz, OAZ or Zfp423) as a component critically required for retinoic acid (RA)-induced differentiation. ZNF423 associates with the RARα/RXRα nuclear receptor complex and is essential for transactivation in response to retinoids. Down-regulation of ZNF423 exp...
ORGANISM(S): Homo sapiens 
2009-04-29 | GSE14627 | GEO
UnknownZNF423: A New Player in Estrogen Receptor-Positive Breast Cancer.
Not available
S-EPMC5968090 | biostudies-literature
GenomicsA novel gene fusion RUNX1/ZNF423 promotes leukemic relapse of NUP98-rearranged AML
A novel gene fusion RUNX1/ZNF423 promotes leukemic relapse of NUP98-rearranged AML
PRJNA917704 | ENA
UnknownGenome-wide interference of ZNF423 with B-lineage transcriptional circuitries in acute lymphoblastic leukemia.
Not available
S-EPMC7948270 | biostudies-literature
TranscriptomicsTranscriptome profiling of NF1 MPNST line RHT-172 after ZNF423 siRNA knockdown
Malignant peripheral nerve sheath tumors (MPNST) are aggressive sarcomas and the leading cause of mortality in neurofibromatosis type 1 (NF1), with limited systemic therapies. Malignant progression can reactivate developmental precursor programs that are largely absent from normal nerve and benign t...
ORGANISM(S): Homo sapiens 
2026-09-11 | GSE320236 | GEO
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