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GenomicsDEGs resulting from activating L-type calcium channel, CaV1.2, in mouse valvular interstitial cells (mVICs).
DEGs resulting from activating L-type calcium channel, CaV1.2, in mouse valvular interstitial cells (mVICs).
PRJNA636195 | ENA
GenomicsLeukemic Cells Hijack Stromal Bioelectricity to Reprogram the Bone Marrow Niche via CaV1.2-Dependent Mechanisms
Leukemic Cells Hijack Stromal Bioelectricity to Reprogram the Bone Marrow Niche via CaV1.2-Dependent Mechanisms
PRJNA1045582 | ENA
ProteomicsQuality control at the cell surface prevents accumulation of damaged L-type CaV1.2 calcium channels
Defective ion channel turnover and clearance of damaged proteins are associated with aging and neurodegeneration. The L-type CaV1.2 voltage-gated calcium channel mediate depolarization-induced calcium signals in heart and brain. Here, we determined the interaction surface between the L-type calcium ...
ORGANISM(S): Oryctolagus cuniculus (Rabbit) Escherichia coli 
2024-02-16 | PXD044630 | Pride
Calcium channel β-subunit Hotspots Cross-linking mass spectrometry Protein-protein docking Cav1.2 surface expression Actin filaments 
ProteomicsQuality control at the cell surface prevents accumulation of damaged L-type CaV1.2 calcium channels
Defective ion channel turnover and clearance of damaged proteins are associated with aging and neurodegeneration. The L-type CaV1.2 voltage-gated calcium channel mediate depolarization-induced calcium signals in heart and brain. Here, we determined the interaction surface between the L-type calcium ...
ORGANISM(S): Oryctolagus cuniculus (Rabbit) Escherichia coli 
2024-02-16 | PXD044723 | Pride
Calcium channel β-subunit Hotspots Cross-linking mass spectrometry Protein-protein docking Cav1.2 surface expression Actin filaments 
TranscriptomicsLeukemic Cells Hijack Stromal Bioelectricity to Reprogram the Bone Marrow Niche via CaV1.2-Dependent Mechanisms
Mesenchymal stromal cells (MSCs) are key components of the tumor microenvironment (TME), influencing leukemia progression through poorly understood mechanisms. Here, the bioelectrical properties of MSCs derived from pediatric acute myeloid leukemia (AML) patients (AML-MSCs) are investigated, identif...
ORGANISM(S): Homo sapiens 
2025-11-28 | GSE248681 | GEO
TranscriptomicsDEGs resulting from activating L-type calcium channel, CaV1.2, in mouse valvular interstitial cells (mVICs).
We report differentially expressed genes as a result of activation of L-type calcium channel CaV1.2 with GOF mutation in mVICs.
ORGANISM(S): Mus musculus 
2021-06-01 | GSE151520 | GEO
Genomics
CaV1.2 from rat white fat cells
PRJNA752806 | ENA
UnknownCav1.2, cell proliferation, and new target in atherosclerosis.
Not available
S-EPMC4392978 | biostudies-literature
UnknownIncreased Ca2+ influx through CaV1.2 drives aortic valve calcification.
Not available
S-EPMC8983132 | biostudies-literature
UnknownRegulation of Cardiac Cav1.2 Channels by Calmodulin.
Not available
S-EPMC10094977 | biostudies-literature
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