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Max Planck Institute of Psychiatry, Munich, Germany Biomedical Center, Molecular Biology, Ludwig-Maximilians-Universitat, Munich, Germany
(1)
Tags
xref:PubMed:35178390
(1)
xref:PubMed:39966398
(1)
xref:PubMed:26116661
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xref:PubMed:39938686
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Mutations in DCHS1 cause mitral valve prolapse.
Not available
S-EPMC4720389
|
biostudies-literature
Cite
Dchs1-Fat4 regulation of osteogenic differentiation in mouse.
Not available
S-EPMC7376788
|
biostudies-literature
Cite
Regulation of neuronal migration by Dchs1-Fat4 planar cell polarity.
Not available
S-EPMC4193925
|
biostudies-literature
Cite
Fat4/Dchs1 signaling between stromal and cap mesenchyme cells influences nephrogenesis and ureteric bud branching.
Not available
S-EPMC4529033
|
biostudies-literature
Cite
DCHS1
DNA copy number loss associated with pediatric urinary tract infection risk.
Not available
S-EPMC7491237
|
biostudies-literature
Cite
Dynamic Expression and Functional Implications of the Cell Polarity Gene, Dchs1, During Cardiac Development.
Not available
S-EPMC12153708
|
biostudies-literature
Cite
DCHS1 Modulates Forebrain Proportions in Modern Humans via a Glycosylation Change.
Not available
S-EPMC12132387
|
biostudies-literature
Cite
Deleterious variants in DCHS1 are prevalent in sporadic cases of mitral valve prolapse.
Not available
S-EPMC5823682
|
biostudies-literature
Cite
Connexin 43 dephosphorylation mediates the Dchs1/YAP/TEAD signaling pathway to impact cardiac fibrosis
Connexin 43 dephosphorylation mediates the Dchs1/YAP/TEAD signaling pathway to impact cardiac fibrosis
PRJNA1048890
|
ENA
Cite
DCHS1, Lix1L, and the Septin Cytoskeleton: Molecular and Developmental Etiology of Mitral Valve Prolapse.
Not available
S-EPMC8874669
|
biostudies-literature
Cite
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