Dsbs - OmicsDI

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Division of Cancer Epigenomics, German Cancer Research Center (DKFZ), INF280, 69120 Heidelberg, Germany (2)
Dept. of Molecular Biosciences, University of Texas at Austin, Texas 78712 USA (2)
Proteomics program, Novo Nordisk Foundation Center for Protein Research, Faculty of Health and Medical Sciences, University of Copenhagen, Blegdamsvej 3B, 2200 Copenhagen, Denmark (2)
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Goethe University Frankfurt, University Hospital, Clinic of Neurology, Exp. Neurology, Heinrich Hoffmann Str. 7, 60590 Frankfurt am Main, Germany (1)
Research Institute of Molecular Pathology (IMP), Campus-Vienna-Biocenter-1, Vienna Biocenter, 1030 Vienna, Austria (1)
Institute of Biomedical Sciences, Academia Sinica, Taipei, Taiwan (1)
Genome Damage and Stability Centre, School of Life Sciences, University of Sussex, Falmer, Brighton, BN1 9RQ, United Kingdom (1)
Lunenfeld-Tanenbaum Research Institute and Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1X5, Canada (1)
Proteomics Facility, The Netherlands Cancer Institute, Plesmanlaan 121, 1066 CX Amsterdam, The Netherlands Biomolecular Mass Spectrometry and Proteomics, Utrecht Institute for Pharmaceutical Sciences, University of Utrecht, Padualaan 8, 3584 CH Utrecht, The Netherlands (1)
Cell and Chemical Biology Leiden University Medical Center Leiden the Netherlands (1)
Institute of Molecular Biology, Ackermannweg 4, 55128 Mainz (1)
Institut de recherches cliniques de Montréal, Center for Genetic and Neurological Diseases,Montréal, QC, Canada Département de biochimie et médecine moléculaire, Université de Montréal, Montréal, QC, Canada Division of Experimental Medicine, McGill University, Montreal, QC, Canada (1)
Department of Human Genetics, University of Miami, Miller School of Medicine, Sylvester Comprehensive Cancer Center, Miami, FL, USA (1)
Institute of Physiological Chemistry, Medical Faculty of TU Dresden, Fiedlerstrasse 42 01307 Dresden, Germany (1)
RC Weatherall Institute of Molecular Medicine, University of Oxford, John Radcliffe Hospital, Headington, Oxford OX3 9DS 2Edinburgh Cancer Research, CRUK Scotland Centre, Institute of Genetics and Cancer, University of Edinburgh, Crewe Road South, EH4 2XU, Edinburgh, UK (1)
Department of Genome Engineering, Institute of Bioorganic Chemistry Polish Academy of Sciences, Poznan, 61-704, Poland (1)
Department of Molecular Genetics and Cell Biology, The University of Chicago, Chicago IL, USA (1)
Genome Integrity and DNA Repair Core Group, Hungarian Centre of Excellence for Molecular Medicine (HCEMM), Szeged, H-6728, Hungary (1)
functionla proteomics, ICR (1)
Department of Environmental and Biological Sciences, University of Tuscia, Viterbo, Italy (1)
The David and Inez Myers Laboratory of Cancer Genetics, Department of Human Molecular Genetics and Biochemistry, Tel Aviv University School of Medicine, Tel Aviv 69987, Israel. (1)
Department of Biological Sciences, Ulsan National Institute of Science and Technology,50 UNIST-gil, Ulju-gun, Ulsan 44919, Republic of Korea (1)
Institute of Aerospace Medicine German Aerospace Center (DLR) Linder Höhe 51147 Cologne, Germany Phone: +49-2203-6011-277 Email: boris.pfander@dlr.de & Institute for Genome Stability in Ageing and Disease University of Cologne CECAD Research Center Joseph-Stelzmann-Str. 26 50931 Cologne, Germany Email: bpfander@uni-koeln.de & Max-Planck Institute of Biochemistry, Am Klopferspitz 18 82152 Martinsried, Germany Phone:+49-89-8578-3050 Fax: +49-89-8578-2454, Email: bpfander@biochem.mpg.de (1)
Dr. Shilpee Dutt, Ph.D., Principal Investigator, Scientific Officer 'G', TMC-ACTREC (1)
DNA damage responses and cancer lab Department of Human Genetics Leiden University Medical Center Eithovenweg 20 2333ZC Leiden The Netherlands (1)
Faculty of Biology, Technion Israel Institute of Technology (1)
Erasmus MC, Rotterdam - NL (1)
junzh128@szu.edu.cn (1)

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Hot spots of DNA double-strand breaks and genomic contacts of human rDNA units are involved in epigenetic regulation [DSBs]

Eukaryotic chromosomes are subjected to spontaneous fragmentation even under quick isolation of DNA in a solid phase by strong treatment with 0.1 M EDTA, 1% SDS and proteinase K (1 mg/ml). The long DNA fragments of excised chromosomal DNA were denoted as forum domains. Mostly forum domains are of 50...

ORGANISM(S): Homo sapiens

2014-10-04 | E-GEOD-49302 | biostudies-arrayexpress

Genome-wide analysis of DSBs sites in HEK293T cells

PRJNA833124 | ENA

DEtail-seq profiled meiotic DSBs in Saccharomyces cerevisiae

PRJNA645445 | ENA

DEtail-seq profiled meiotic DSBs in Mus musculus

PRJNA645811 | ENA

DEtail-seq profiled meiotic DSBs in Homo sapiens

PRJNA645446 | ENA

Detecting non-canonically-targeted SpCas9 generated DSBs in human cells by LAM-HTGTS

PRJNA791602 | ENA

Meiotic DNA double strand breaks in the yeast Saccaromyces cerevisiae

DNA double-strand breaks (DSBs) initiate meiotic recombination. Past DSB-mapping studies have used rad50S or sae2? mutants, which are defective in break processing, to accumulate DSBs, and report large (= 50 kb) “DSB-hot” regions that are separated by “DSB-cold” domains of similar size. Substantial ...

ORGANISM(S): Saccharomyces cerevisiae

2007-12-30 | E-GEOD-8981 | biostudies-arrayexpress

Cohesin profiling at DSBs

Chromatin undergoes major remodeling around DNA double strand breaks (DSB) to promote repair and DNA damage response (DDR) activation. We recently reported a high resolution map of gammaH2AX around multiple breaks on the human genome, using a new cell-based DSB inducible system. In an attempt to fur...

ORGANISM(S): Homo sapiens

2012-01-01 | E-TABM-1164 | biostudies-arrayexpress

R-loops and Topoisomerase 1 facilitate formation of transcriptional DSBs at gene bodies of hypertranscribed cancer genes

PRJNA1007657 | ENA

A POLD3/BLM dependent pathway handles DSBs in transcribed chromatin upon excessive RNA:DNA hybrids accumulation

PRJEB51910 | ENA