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Department of Medical Biochemistry and Biophysics, Karolinska Institutet, Sweden Centre for Inherited Metabolic Diseases, Karolinska University Hospital, Sweden
(1)
Harry Perkins Institute of Medical Research and ARC Centre of Excellence in Synthetic Biology, QEII Medical Centre, Nedlands, Western Australia 6009, Australia
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Harry Perkins Institute of Medical Research and Centre of Excellence in Synthetic Biology, QEII Medical Centre, Nedlands, Western Australia 6009, Australia
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Tags
xref:PubMed:26501111
(6)
xref:PubMed:37093546
(4)
xref:PubMed:30126926
(3)
xref:PubMed:42338969
(1)
xref:PubMed:37889747
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xref:PubMed:41237252
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ELAC2/RNaseZ-linked cardiac hypertrophy in Drosophila melanogaster.
Not available
S-EPMC8419712
|
biostudies-literature
Cite
Structural insights into human ELAC2 as a tRNA 3' processing enzyme.
Not available
S-EPMC11602120
|
biostudies-literature
Cite
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (miRNA-Seq)
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (miRNA-Seq)
PRJNA828612
|
ENA
Cite
Association of HPC2/ELAC2 genotypes and prostate cancer.
Not available
S-EPMC1287872
|
biostudies-literature
Cite
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (RNA-Seq II)
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (RNA-Seq II)
PRJNA828608
|
ENA
Cite
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (RNA-Seq I)
ELAC2 is a prostate cancer rheostat that regulates nuclear and mitochondrial RNA metabolism (RNA-Seq I)
PRJNA828610
|
ENA
Cite
Nuclear ELAC2 overexpression is associated with increased hazard for relapse after radical prostatectomy.
Not available
S-EPMC6697635
|
biostudies-literature
Cite
Mutations in ELAC2 associated with hypertrophic cardiomyopathy impair mitochondrial tRNA 3'-end processing.
Not available
S-EPMC6764886
|
biostudies-literature
Cite
A homozygous splicing mutation in ELAC2 suggests phenotypic variability including intellectual disability with minimal cardiac involvement.
Not available
S-EPMC5073853
|
biostudies-literature
Cite
ELAC2 mutations cause a mitochondrial RNA processing defect associated with hypertrophic cardiomyopathy.
Not available
S-EPMC3738821
|
biostudies-literature
Cite
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