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UnknownAMPK activator AICAR ameliorates ischaemia reperfusion injury in the rat kidney.
Not available
S-EPMC3402813 | biostudies-literature
UnknownDexmedetomidine preconditioning ameliorates kidney ischemia-reperfusion injury.
Not available
S-EPMC4186414 | biostudies-literature
UnknownCharacterizing the Key Metabolic Pathways of the Neonatal Mouse Heart Using a Quantitative Combinatorial Omics Approach.
Not available
S-EPMC5904546 | biostudies-literature
UnknownAltered acylcarnitine metabolism and inflexible mitochondrial fuel utilization characterize the loss of neonatal myocardial regeneration capacity.
Not available
S-EPMC10167339 | biostudies-literature
UnknownDiscovery and characterization of ORM-11372, a novel inhibitor of the sodium-calcium exchanger with positive inotropic activity.
Not available
S-EPMC7707092 | biostudies-literature
ProteomicsAccumulation of acylcarnitines and mitochondrial dysfunction characterize the loss of neonatal myocardial regeneration capacity
Myocardial regeneration capacity declines during the first week after birth and is linked to the adaptation to oxidative metabolism. Utilizing this regenerative window, we characterized the metabolic changes in myocardial injury in 1-day-old regeneration-competent and 7-day-old regeneration-compromi...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
2022-02-04 | MSV000088783 | MassIVE
MetabolomicsGNPS - Accumulation of acylcarnitines and mitochondrial dysfunction characterize the loss of neonatal myocardial regeneration capacity
Myocardial regeneration capacity declines during the first week after birth and is linked to the adaptation to oxidative metabolism. Utilizing this regenerative window, we characterized the metabolic changes in myocardial injury in 1-day-old regeneration-competent and 7-day-old regeneration-compromi...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
2022-02-04 | MSV000088783 | GNPS
TranscriptomicsBone marrow-derived stromal cells from double transgenic rats (of Sprague-Dawley origin) harboring the human angiotensinogen and human renin genes, and control rats
Hypertension and persistent activation of the renin-angiotensin system (RAS) are predisposing factors for development of acute kidney injury (AKI). Although bone marrow-derived stromal cells (BMSCs) have shown therapeutic promise in treatment of AKI, the impact of pathological RAS on BMSC functional...
ORGANISM(S): Rattus norvegicus 
2014-03-31 | E-GEOD-51593 | biostudies-arrayexpress
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