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TranscriptomicsG0S2 functions as a tumor supressor through represssion of Myc signaling pathways
Comparions on gene expression in murine embyonic fibroblasts (MEFs) wild-type vs G0S2 null MEFs Total RNA obtained from spontanously immortalized MEF cell line E2 from wild-type C57BL/6 mice (E2) and spontaneously immortalized MEF cell line E6 from G0S2 knockout C57BL/6 mice; MEF lines were used at ...
ORGANISM(S): Mus musculus 
2016-06-01 | E-GEOD-74696 | biostudies-arrayexpress
TranscriptomicsInfluence of G0s2 gene knockdown on primary hepatocyte
Hepatocellular carcinoma is the third leading cause of cancer death worldwide, and it is necessary to elucidate the mechanism of hepatocarcinogenesis. Circadian clock systems have been related in cell cycle and carcinogenesis. In this study, hepatocarcinogenesis induced by DEN administration in mice...
ORGANISM(S): Mus musculus 
2013-08-01 | E-GEOD-39288 | biostudies-arrayexpress
GenomicsEffect of G0s2 and Rgs16 knockdown on hepatic transcriptome
Effect of G0s2 and Rgs16 knockdown on hepatic transcriptome
PRJNA751237 | ENA
GenomicsG0S2 modulates normal vitreous-induced proliferation in endothelial cells [1]
G0S2 modulates normal vitreous-induced proliferation in endothelial cells [1]
PRJNA1221902 | ENA
GenomicsG0S2 modulates normal vitreous-induced proliferation in endothelial cells [2]
G0S2 modulates normal vitreous-induced proliferation in endothelial cells [2]
PRJNA1221904 | ENA
GenomicsG0S2 promotes antiestrogenic and pro-migratory responses in ER+ and ER- breast cancer cells.
G0S2 promotes antiestrogenic and pro-migratory responses in ER+ and ER- breast cancer cells.
PRJNA946669 | ENA
TranscriptomicsInfluence of G0s2 gene knockdown on primary hepatocyte
Hepatocellular carcinoma is the third leading cause of cancer death worldwide, and it is necessary to elucidate the mechanism of hepatocarcinogenesis. Circadian clock systems have been related in cell cycle and carcinogenesis. In this study, hepatocarcinogenesis induced by DEN administration in mice...
ORGANISM(S): Mus musculus 
2013-08-01 | GSE39288 | GEO
TranscriptomicsThe inhibition of the lipolytic inhibitor G0s2 enhancers affects lipid metabolism
The G0/G1 switch gene 2 (G0s2) is a selective inhibitor of adipose triglyceride lipase (ATGL) which is the rate-limiting enzyme for triglycerides (TGs) hydrolysis in adipocytes, and regulates the mobilization of TGs in adipocytes and hepatocytes. The expression and functional disorders of G0S2 are a...
ORGANISM(S): Mus musculus 
2026-10-07 | GSE278930 | GEO
TranscriptomicsG0S2 promotes antiestrogenic and pro-migratory responses in ER+ and ER- breast cancer cells.
G0/G1 switch gene 2 (G0S2) is known to inhibit lipolysis through inhibition of adipose triglyceride lipase (ATGL). Although G0S2 has also been proposed to be involved in cancer progression, this role appears complex, and mechanisms are unclear. We recently reported a role for G0S2 in ER+ breast canc...
ORGANISM(S): Homo sapiens 
2023-04-11 | GSE227698 | GEO
TranscriptomicsG0S2 modulates normal vitreous-induced proliferation in endothelial cells [2]
Abnormal blood vessel growth in the eye is a leading cause of vision loss globally, particularly in diseases like diabetic retinopathy where the vitreous plays a crucial but poorly understood role in disease progression. While we know the vitreous can stimulate blood vessel growth, the specific mole...
ORGANISM(S): Homo sapiens 
2025-03-03 | GSE289206 | GEO
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