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pride (587)
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Mass Spectrometry (587)
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Instrument Platform

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Illumina NovaSeq 6000 (19)
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MS:1000031 (3)
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Publication Date

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UNIPROT ID/AC

P24871 (1)
P40957 (1)
Q9Y2T1 (1)
P24870 (1)
O14640 (1)
P24865 (1)
P78972 (1)
O13286 (1)
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P42574 (1)
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P49841 (1)
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P40380 (1)

ENSEMBL ID

ENSG00000139318 (1)
ENSG00000168646 (1)

CHEBI ID

CHEBI:16027 (20)
CHEBI:17754 (15)
CHEBI:14314 (14)
CHEBI:17234 (13)
CHEBI:16761 (5)
CHEBI:16905 (4)
CHEBI:15422 (4)
CHEBI:28087 (3)
CHEBI:29108 (3)
CHEBI:16236 (3)
CHEBI:17665 (3)
CHEBI:16908 (3)
CHEBI:18021 (3)
CHEBI:27082 (2)
CHEBI:29052 (2)
CHEBI:28602 (2)
CHEBI:30031 (2)
CHEBI:15343 (2)
CHEBI:15361 (2)
CHEBI:17835 (2)
CHEBI:17794 (2)
CHEBI:16001 (2)
CHEBI:16526 (2)
CHEBI:15846 (2)
CHEBI:16108 (2)
CHEBI:22204 (1)
CHEBI:63836 (1)
CHEBI:15625 (1)
CHEBI:17312 (1)
CHEBI:4167 (1)

Metabolite Name

Adenine (31)
Uridine (28)
Hypoxanthine (27)
Adenosine (26)
Uracil (24)
Citric acid (24)
Inosine (24)
L-Tyrosine (23)
Uric acid (21)
L-Tryptophan (20)
Azelaic acid (20)
Guanine (19)
Creatinine (19)
L-Phenylalanine (19)
Kynurenic acid (18)
Pantothenic acid (18)
Taurine (18)
Stearic acid (18)
Guanosine (18)
L-Arginine (17)
Fumaric acid (17)
Arachidonic acid (17)
Palmitic acid (17)
Xanthine (17)
Thymine (16)
L-Threonine (16)
Glyceric acid (16)
Malic acid (16)
Linoleic acid (16)
Cytidine (16)

First Public Date

2014 (1)

Modelling Approach

Ordinary differential equation model (25)

Lab affiliation

Department of Immunology, College of Basic Medical Science, China Medical University, Shenyang, Liaoning 110122, China (4)
Fraunhofer Institute for Molecular Biology and Applied Ecology IME Auf dem Aberg 1 | 57392 Schmallenberg | Germany (4)
State Key Laboratory of Biocatalysis and Enzyme Engineering, School of Life Sciences, Hubei University, Wuhan,Hubei 430062, China (4)
Amsterdam UMC, Vrije Universiteit Amsterdam, Medical Oncology, Cancer Center Amsterdam, OncoProteomics Laboratory, Amsterdam, Netherlands (3)
Beijing proteome research center (3)
Department of Plant Systems Biology, University of Hohenheim, 70599 Stuttgart, Germany (3)
Institute For Systems Biology (3)
Institute of Functional Epigenetics, Helmholtz Zentrum München, 85764 Neuherberg, Germany. (3)
Lanzhou University (3)
Max Planck Institute for Terrestrial Microbiology Karl-von-Frisch Str. 10 35043 Marburg Germany (3)
Research Centre for Genetic Engineering and Biotechnology “Georgi D Efremov”, Macedonian Academy of Sciences and Arts, Skopje, Krste Misirkov 2, 1000 Skopje, Republic of North Macedonia (3)
The Institute of Cancer Research (3)
Brain Injury Center, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Head Trauma, Shanghai, China, 200127. (2)
Clinical Protein Science & Imaging, Dept. of Measurement Technology and Industrial Electrical Engineering, Lund University, Lund, Sweden (2)
College of Life Sciences, South-Central University for Nationalities (2)
DNRF Center for Gene Expression Department of Cellular and Molecular Medicine Blegdamsvej 3B University of Copenhagen 2200 Copenhagen N, Denmark (2)
Department of Analytical Chemistry and CAS Key Laboratory of Receptor Research, Shanghai Institute of Materia Medica, Chinese Academy of Sciences (2)
Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, MA 02215, USA Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA (2)
Department of Clinical Laboratory, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, China. The Key Laboratory of Molecular Biology of Infectious Diseases designated by the Chinese Ministry of Education, Chongqing Medical University, Chongqing, China. (2)
Department of Pathophysiology, Key Laboratory of Ministry of Education for Neurological Disorders, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology (2)
Fraunhofer Attract Eco’n’OMICs, Fraunhofer Institute for Molecular Biology and Applied Ecology IME, Auf dem Aberg 1, 57392 Schmallenberg. (2)
Fudan University (2)
Howard Hughes Medical Institute Beth Israel Deaconess Medical Center Harvard Medical School (2)
Huazhong University of Science and Technology (2)
Institute for Cancer Research, Shenzhen Bay Laboratory, China. (2)
Institute for Systems Biology (2)
Institute of Computing Technology, CAS (2)
Institute of Drug Discovery Technology, Ningbo University, Ningbo, Zhejiang, 315211 China (2)
Lanzhou University, Lanzhou, Gansu, China (2)
Lee Kong Chian School of Medicine, School of Biological Sciences, Nanyang Technological University, Singapore (2)

IsPrivate

False (25)

Model Flag

Sbml Extended (1)

Modification

4-tetrahydrothiazine-3-carboxylic acid (2)
N6 (1)
Not available (1)

Study type

Transcription profiling by array (163)
RNA-seq of coding RNA (26)
ChIP-seq (20)
Methylation profiling by high throughput sequencing (15)
RNA-seq of coding RNA from single cells (12)
Comparative genomic hybridization by array (9)
ChIP-chip by tiling array (6)
Other (6)
Unknown experiment type (5)
DNA-seq (4)
RNA-seq of non coding RNA (4)
Methylation profiling by array (3)
Cell line - Single-cell sequencing (2)
RNAi profiling by array (2)
Spatial transcriptomics by high-throughput sequencing (2)
MicroRNA profiling by array (2)
Cell line - One-color microarray (1)
High-throughput sequencing (1)
Transcription profiling by tiling array (1)
Hi-C (1)

Release Date

2022 (112)
2025 (97)
2024 (95)
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2020 (69)
2014 (57)
2010 (57)
2016 (49)
2015 (48)
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2011 (37)
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2009 (29)
2008 (27)
2026 (23)
2007 (8)
2006 (3)
2004 (2)
2000 (2)
2005 (1)
2003 (1)
2002 (1)
2001 (1)

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GPR109a-AMPK Axis Mediates the Attenuation of Uric Acid-Induced M1 Macrophage Polarization by β-hydroxybutyrate from Lacticaseibacillus rhamnosus M2b

Hyperuricemia (HUA) is a metabolic disorder marked by increased blood uric acid (UA) levels, closely linked to conditions such as gout. UA-induced macrophage M1 polarization fundamentally exacerbates inflammatory pathophysiology, but current HUA-specific immunoregulatory treatments are inadequat...

2026-01-26 | MTBLS13224 | MetaboLights

Integrated Multi-omics Elucidation of the Uric Acid Degradation Pathway in Limosilactobacillus fermentum M5e

Hyperuricemia is a growing global health concern associated with gout, renal diseases, and cardiovascular disorders. Current pharmacotherapies are often limited by side effects, underscoring the need for alternative strategies. In this study, we isolated Limosilactobacillus fermentum M5...

2026-01-26 | MTBLS13223 | MetaboLights

pH stress in Desulfovibrio vulgaris Hildenborough

Using data from microarray experiments, we investigated the effects of excess NaCl on D. vulgaris. Keywords: stress response, time course Comparison of cells treated with pH10 pH to untreated cells at times of 0,120,240,30,60 min.

ORGANISM(S): Desulfovibrio vulgaris str. Hildenborough

2007-08-31 | E-GEOD-8197 | biostudies-arrayexpress

Cell wide response to air exposure in Desulfovibrio vulgaris Hildenborough

We investigated the effects of air exposure on Desulfovibrio vulgaris Hildenborough using microarrays. Keywords: stress response, time course Comparison of cells treated with air to untreated cells at 0,10,30,120,240 min.

ORGANISM(S): Desulfovibrio vulgaris str. Hildenborough

2007-08-08 | E-GEOD-8196 | biostudies-arrayexpress

Cell wide response to low oxygen exposure in Desulfovibrio vulgaris Hildenborough

We investigated the effects of low level oxygen on Desulfovibrio vulgaris Hildenborough using microarrays. Keywords: time course, stress response Comparison of cells treated with 1000 ppm oxygen to untreated cells at 60,120,240 min.

ORGANISM(S): Desulfovibrio vulgaris str. Hildenborough

2010-06-10 | E-GEOD-7816 | biostudies-arrayexpress

Gene expression of rat cerebellum in a new animal model of hepatic encephalopathy (HE)

Identify differentially expressed genes related to the neurodegenerative process in a new animal model of hepatic encephalopathy (HE). The animal model consists on the simulation of several bouts of HE in PCA rats, being the precipitant factors of the episodes ammonia (NH3) and/or lipopolisaccharide...

ORGANISM(S): Rattus norvegicus

2015-07-29 | E-GEOD-71425 | biostudies-arrayexpress

Salt/NaCl stress in Desulfovibrio vulgaris Hildenborough

This set of microarray data was used to investigate the effects of D. vulgaris adaptation to excess NaCl. Keywords: stress response, one time point (the mid-log phase for both control and treated cells) Comparison of cells treated with 250 Salt:NaCl to untreated cells at times of mid-log phase min.

ORGANISM(S): Desulfovibrio vulgaris str. Hildenborough

2010-04-11 | E-GEOD-14343 | biostudies-arrayexpress

Energy metabolism pathways of the sulfate-reducer Desulfovibrio vulgaris Hildenborough

This SuperSeries is composed of the following subset Series: GSE8015: Pyruvate fermentation vs Lactate-Sulfate GSE8037: Hydrogen vs Lactate as electron donor in Sulfate reduction GSE8071: Pyruvate vs Lactate as electron donor in Sulfate reduction GSE8072: Thiosulfate vs Sulfate as electron acceptor ...

ORGANISM(S): Desulfovibrio vulgaris subsp. vulgaris str. Hildenborough

2008-04-05 | E-GEOD-8069 | biostudies-arrayexpress

Pyruvate vs Lactate as electron donor in Sulfate reduction

In order to obtain a global view of energy metabolism pathways of the sulfate-reducer Desulfovibrio vulgaris Hildenborough and the proteins involved therein whole-genome microarrays were used to compare the transcriptional response of cells grown with hydrogen/sulfate, pyruvate/sulfate, lactate/thio...

ORGANISM(S): Desulfovibrio vulgaris subsp. vulgaris str. Hildenborough

2008-05-31 | E-GEOD-8071 | biostudies-arrayexpress

Thiosulfate vs Sulfate as electron acceptor in Sulfate reduction

ORGANISM(S): Desulfovibrio vulgaris subsp. vulgaris str. Hildenborough

2008-05-31 | E-GEOD-8072 | biostudies-arrayexpress

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