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UnknownHemidesmosome Mutations Contribute to the Onset and Severity of Acquired Autoimmune Bullous Diseases.
Not available
S-EPMC12906664 | biostudies-literature
UnknownKeratinocyte-Specific Peptide-Based Surfaces for Hemidesmosome Upregulation and Prevention of Bacterial Colonization.
Not available
S-EPMC7494210 | biostudies-literature
UnknownTargeted proteolysis of plectin isoform 1a accounts for hemidesmosome dysfunction in mice mimicking the dominant skin blistering disease EBS-Ogna.
Not available
S-EPMC3228830 | biostudies-literature
UnknownCytoplasmic plaque formation in hemidesmosome development is dependent on SoxF transcription factor function.
Not available
S-EPMC3433475 | biostudies-literature
UnknownPhosphorylation of threonine 1736 in the C-terminal tail of integrin β4 contributes to hemidesmosome disassembly.
Not available
S-EPMC3327322 | biostudies-literature
UnknownDynamic relationship of focal contacts and hemidesmosome protein complexes in live cells.
Not available
S-EPMC2882194 | biostudies-literature
UnknownInteractions of laminin β3 fragment with β1-integrin receptor: A revisit of the apical ectoplasmic specialization-blood-testis-barrier-hemidesmosome functional axis in the testis.
Not available
S-EPMC3271660 | biostudies-literature
UnknownAdenovirus-Mediated LAMA3 Transduction Enhances Hemidesmosome Formation and Periodontal Reattachment during Wound Healing.
Not available
S-EPMC7334303 | biostudies-literature
no omics definedTranscription profiling of human keratinocytes treated with JNK inhibitor vs. control reveals inhibition of JNK in keratinocyte cultures in vitro induces virtually all aspects of epidermal differentiation in vivo
In inflamed tissue, normal signal transduction pathways are altered by extracellular signals. For example, the JNK pathway is activated in psoriatic skin, which makes it an attractive target for treatment. To define comprehensively the JNK-regulated genes in human epidermal keratinocytes, we compare...
ORGANISM(S): Homo sapiens 
2007-10-26 | E-GEOD-4828 | biostudies-arrayexpress
UnknownUV exposure modulates hemidesmosome plasticity, contributing to long-term pigmentation in human skin.
Not available
S-EPMC4398603 | biostudies-literature
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